Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Glutamic acid decarboxylase immunoreactivity in the mossy fiber terminals of the hippocampus of genetic absence epileptic rats.

Turkish neurosurgery - 1 Jan 2011

Sirvanci Serap, Canillioglu Yasemin, Akakin Dilek, Midillioglu Sukru, Yildiz Sercan Dogukan, Onat Filiz, San Tangul

Abstract excerpt

AIM: Genetic absence epilepsy rats from Strasbourg (GAERS) provide a model of absence epilepsy. Although excessive GABA mediation within the thalamo-cortico-thalamic circuit has been shown to play a role in absence epilepsy, neuronal networks of hippocampus have recently received attention. Glutamic acid decarboxylase (GAD) was previously shown to be increased after convulsive seizures in the...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Dentate Gyrus
Share to Epilepsy, Absence

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to gamma-Aminobutyric Acid

Share to Glutamate Decarboxylase

Share to Microscopy, Immunoelectron

Share to Neural Inhibition

Share to Presynaptic Terminals

Share to Rats, Mutant Strains

Source

Identifiers

PMID: 22194107
DOI: 10.5137/1019-5149.JTN .4353-11.1

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Glutamic acid decarboxylase immunoreactivity in the mossy fiber terminals of the hippocampus of genetic absence epileptic rats.PMID 22194107Read ↗

Ultrastructural GABA immunocytochemistry in the mossy fiber terminals of Wistar and genetic absence epileptic rats receiving amygdaloid kindling stimulations.Semantic proximity Read ↗

Astrocytes may play a role in the etiology of absence epilepsy: a comparison between immature GAERS not yet expressing seizures and adults.Semantic proximity Read ↗

Audiogenic kindling activates glutamatergic system in the hippocampus of rats with genetic predisposition to audiogenic seizures.Semantic proximity Read ↗

Maladaptive myelination promotes epileptogenesis in absence epilepsySemantic proximity Read ↗

Low distribution of synaptic vesicle protein 2A and synaptotagimin-1 in the cerebral cortex and hippocampus of spontaneously epileptic rats exhibiting both tonic convulsion and absence seizure.Semantic proximity Read ↗

Seizure expression, behavior, and brain morphology differences in colonies of Genetic Absence Epilepsy Rats from Strasbourg.Semantic proximity Read ↗

Increased inhibitory synaptic activity in the hippocampus (CA1) of genetic absence epilepsy rats: Relevance of kindling resistance.Semantic proximity Read ↗

Susceptibility to focal and generalized seizures in Wistar rats with genetic absence-like epilepsy.Semantic proximity Read ↗

Experimental Models of Absence EpilepsySemantic proximity Read ↗

[Cytomorphological features of the neocortex in rats with genetic predisposition to absence epilepsy].Semantic proximity Read ↗

Epilepsy in mice deficient in the 65-kDa isoform of glutamic acid decarboxylase.Semantic proximity Read ↗

Increased expression of mRNA encoding ferritin heavy chain in brain structures of a rat model of absence epilepsy.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.