Immunophenotype of vitamin D receptor polymorphism associated to risk of HIV-1 infection and rate of disease progression.

doi:10.2174/157016210793499330

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Community

Back to search

Article

Immunophenotype of vitamin D receptor polymorphism associated to risk of HIV-1 infection and rate of disease progression.

Current HIV research - 1 Sept 2010

Torres Carmen, Sánchez de la Torre Manuel, García-Moruja Carelia, Carrero Antonio José, Trujillo María del Monte, Fibla Joan, Caruz Antonio

Abstract excerpt

Vitamin-D-receptor (VDR) mediates immunomodulatory effects of vitamin-D₃ (VD₃). The VDR-rs1544410_GG polymorphism has been associated with delayed progression rates to AIDS and resistance to HIV-1 infection. The aim of the present study was to investigate differences in VD₃ mediated effects on rs1544410 genotyped dendritic cells (DCs) and macrophages (MDM), key cells involved in HIV-1 infection. Immature DCs...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cholecalciferol

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Dendritic Cells

Share to Genes, Reporter

Share to HIV Long Terminal Repeat

Share to Immunophenotyping

Share to Macrophages

Share to Receptors, Calcitriol

Share to Spain

Source

Open in PubMed

Identifiers

PMID: 20642435
DOI: 10.2174/157016210793499330

Genes

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Immunophenotype of vitamin D receptor polymorphism associated to risk of HIV-1 infection and rate of disease progression.PMID 20642435Read ↗

The association of CD209 promoter variants with tuberculosis infection susceptibility, AIDS development, and treatment response outcomes among an HIV-1 Moroccan populationSemantic proximity Read ↗

Vitamin-D pathway genes and HIV-1 disease progression in injection drug users.Semantic proximity Read ↗

A functional toll-like receptor 8 variant is associated with HIV disease restriction.Semantic proximity Read ↗

The CD14 C-260T single nucleotide polymorphism (SNP) modulates monocyte/macrophage activation in treated HIV-infected individuals.Semantic proximity Read ↗

Polymorphisms in Toll-like receptor 9 influence the clinical course of HIV-1 infection.Semantic proximity Read ↗

Polymorphisms of innate immunity genes influence disease progression in HIV-1-infected children.Semantic proximity Read ↗

Toll-like receptor agonists enhance HIV-specific T cell response mediated by plasmacytoid dendritic cells in diverse HIV-1 disease progression phenotypes.Semantic proximity Read ↗

A FcɣRIIa polymorphism has a HLA-B57 and HLA-B27 independent effect on HIV disease outcome.Semantic proximity Read ↗

Strong Association of the rs4986790 Single Nucleotide Polymorphism (SNP) of the Toll-Like Receptor 4 (TLR4) Gene with Human Immunodeficiency Virus (HIV) Infection: A Meta-Analysis.Semantic proximity Read ↗

An assessment of toll-like receptor 7 and 8 gene polymorphisms with susceptibility to HIV-1 infection, AIDS development and response to antiretroviral therapy.Semantic proximity Read ↗

TLR Agonists Enhance HIV-Specific T-Cell Response Mediated by Plasmacytoid Dendritic Cells in Diverse HIV-1 Disease Progression PhenotypesSemantic proximity Read ↗

Polymorphisms in toll-like receptor 4 and toll-like receptor 9 influence viral load in a seroincident cohort of HIV-1-infected individuals.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.