Deletion of Late Cornified Envelope 3B and 3C genes is not associated with atopic dermatitis.

doi:10.1038/jid.2010.88

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Community

Back to search

Article

Deletion of Late Cornified Envelope 3B and 3C genes is not associated with atopic dermatitis.

The Journal of investigative dermatology - 1 Aug 2010

Bergboer Judith G M, Zeeuwen Patrick L J M, Irvine Alan D, Weidinger Stephan, Giardina Emiliano, Novelli Giuseppe, Den Heijer Martin, Rodriguez Elke, Illig Thomas, Riveira-Munoz Eva, Campbell Linda E, Tyson Jess, Dannhauser Emma N, O'Regan Gráinne M, Galli Elena, Klopp Norman, Koppelman Gerard H, Novak Natalija, Estivill Xavier, McLean W H Irwin, Postma Dirkje S, Armour John A L, Schalkwijk Joost

Abstract excerpt

Atopic dermatitis (AD) and psoriasis are common skin diseases characterized by cutaneous inflammation and disturbed epidermal differentiation. Genome-wide analyses have shown overlapping susceptibility loci, such as the epidermal differentiation complex on chromosome 1q21. Recently, a deletion on 1q21 (LCE3C_LCE3B-del), comprising LCE3B and LCE3C, two members of the late cornified envelope (LCE) gene cluster, was...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Asthma

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Dermatitis, Atopic

Share to Europe

Share to Filaggrin Proteins

Share to Gene Deletion

Share to Immunoglobulin E

Share to Linkage Disequilibrium

Share to White People

Source

Open in PubMed

Identifiers

PMID: 20376060
DOI: 10.1038/jid.2010.88

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Deletion of Late Cornified Envelope 3B and 3C genes is not associated with atopic dermatitis.PMID 20376060Read ↗

Deletion of the late cornified envelope LCE3B and LCE3C genes as a susceptibility factor for psoriasis.Semantic proximity Read ↗

Deletion of the late cornified envelope genes LCE3C and LCE3B is associated with psoriasis in a Chinese population.Semantic proximity Read ↗

Psoriasis-Associated Late Cornified Envelope (LCE) Proteins Have Antibacterial Activity.Semantic proximity Read ↗

Association of the late cornified envelope-3 genes with psoriasis and psoriatic arthritis: a systematic review.Semantic proximity Read ↗

Deletion of late cornified envelope genes, LCE3C_LCE3B-del, is not associated with psoriatic arthritis in Tunisian patients.Semantic proximity Read ↗

Pushing the Envelope in Psoriasis: Late Cornified Envelope Proteins Possess Antimicrobial Activity.Semantic proximity Read ↗

Deletion of LCE3C and LCE3B genes is associated with psoriasis in a northern Chinese population.Semantic proximity Read ↗

Deletion of LCE3C and LCE3B genes at PSORS4 does not contribute to susceptibility to psoriatic arthritis in German patients.Semantic proximity Read ↗

Mutation analysis of the LCE3B/LCE3C genes in Psoriasis.Semantic proximity Read ↗

Replication of LCE3C-LCE3B CNV as a risk factor for psoriasis and analysis of interaction with other genetic risk factors.Semantic proximity Read ↗

Antimicrobial Late Cornified Envelope Proteins: The Psoriasis Risk Factor Deletion of LCE3B/C Genes Affects Microbiota Composition.Semantic proximity Read ↗

Failure to find evidence for deletion of LCE3C and LCE3B genes at PSORS4 contributing to psoriasis susceptibility in Tunisian families.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.