Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Association of FcGRIIa with Graves' disease: a potential role for dysregulated autoantibody clearance in disease onset/progression.

Clinical endocrinology - 1 Jul 2010

Yesmin Kadija, Hargreaves Chantal, Newby Paul R, Brand Oliver J, Heward Joanne M, Franklyn Jayne A, Gough Stephen C L, Simmonds Matthew J

Abstract excerpt

OBJECTIVE: Although autoantibody production is a key feature of autoimmunity, it is not known whether variation in autoantibody production and clearance pathways is involved in disease susceptibility. The Fc Gamma Receptor IIa (FcGRIIa) molecule is involved in the clearance of autoantibodies and...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Autoantibodies
Share to Case-Control Studies
Share to Disease Progression

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Genetic Predisposition to Disease

Share to Graves Disease

Share to Polymorphism, Single Nucleotide

Share to Receptors, IgG

Source

Identifiers

PMID: 20148910
DOI: 10.1111/j.1365-2265.2010.03780.x

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 2 Sept 2026. Prepared record publication-20148910-2026-09-02.2. Manifest publication-graph-cohort-2026-09-02.2.

Source receipt: PubMed via Helena Literature Corpus, retrieved 2 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Association of FcGRIIa with Graves' disease: a potential role for dysregulated autoantibody clearance in disease onset/progression.PMID 20148910Read ↗

Association of Fc receptor-like 5 (FCRL5) with Graves' disease is secondary to the effect of FCRL3.Semantic proximity Read ↗

Contribution of single nucleotide polymorphisms within FCRL3 and MAP3K7IP2 to the pathogenesis of Graves' disease.Semantic proximity Read ↗

The Genetic Basis of Graves DiseaseSemantic proximity Read ↗

Role of genetic and non-genetic factors in the etiology of Graves' disease.Semantic proximity Read ↗

Dysregulated Antibody, Natural Killer Cell and Immune Mediator Profiles in Autoimmune Thyroid DiseasesSemantic proximity Read ↗

Genetic Variation in Low-To-Medium-Affinity Fcγ Receptors: Functional Consequences, Disease Associations, and Opportunities for Personalized MedicineSemantic proximity Read ↗

Tag SNP screening of the PDCD1 gene for association with Graves' disease.Semantic proximity Read ↗

Association of FCGR2A and FCGR2A-FCGR3A haplotypes with susceptibility to giant cell arteritis.Semantic proximity Read ↗

Association of the thyroid stimulating hormone receptor gene (TSHR) with Graves' disease.Semantic proximity Read ↗

Analysis of the Fc receptor-like-3 (FCRL3) locus in Caucasians with autoimmune disorders suggests a complex pattern of disease association.Semantic proximity Read ↗

Genetic analysis in young-age-of-onset Graves' disease reveals new susceptibility loci.Semantic proximity Read ↗

Genetic association of Fc receptor-like glycoprotein with susceptibility to Graves' disease in a Chinese Han population.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.