Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

HIVAN phenotype: consequence of epithelial mesenchymal transdifferentiation.

American journal of physiology. Renal physiology - 1 Mar 2010

Yadav Anju, Vallabu Sridevi, Kumar Dileep, Ding Guohua, Charney Douglas N, Chander Praveen N, Singhal Pravin C

Abstract excerpt

Human immunodeficiency virus (HIV)-1-associated nephropathy (HIVAN) is characterized by proliferation of glomerular and tubular epithelial cells. We studied the role of epithelial mesenchymal transdifferentiation (EMT) in the development of HIVAN phenotype. Renal cortical sections from six FVB/N (control) and six Tg26 (HIVAN) mice were immunolabeled for PCNA, alpha-smooth muscle actin (alpha-SMA),...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Angiotensin II
Share to Angiotensin II Type 1 Receptor Blockers

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Benzoates

Share to Cell Transdifferentiation

Share to Kidney Glomerulus

Share to Kidney Tubules

Share to Podocytes

Share to S100 Proteins

Source

Open in PubMed Open full text

Identifiers

PMID: 20015943
DOI: 10.1152/ajprenal.00415.2009
PMCID: PMC2838599

Genes

PCNA
SRM
ANG
PGC
ZEB2

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 6 Sept 2026. Manifest publication-graph-cohort-2026-09-06.1.

Source receipt: pubmed, retrieved 6 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

HIVAN phenotype: consequence of epithelial mesenchymal transdifferentiation.PMID 20015943Read ↗

HIV-1 Nef induces dedifferentiation of podocytes in vivo: a characteristic feature of HIVAN.Semantic proximity Read ↗

Tubular cell phenotype in HIV-associated nephropathy: role of phospholipid lysophosphatidic acid.Semantic proximity Read ↗

Dysregulation of podocyte phenotype in idiopathic collapsing glomerulopathy and HIV-associated nephropathy.Semantic proximity Read ↗

Human Immunodeficiency Virus-1 Induces Loss of Contact Inhibition in PodocytesSemantic proximity Read ↗

The role of EMT in renal fibrosisSemantic proximity Read ↗

Tubular-specific expression of HIV protein Vpr leads to severe tubulointerstitial damage accompanied by progressive fibrosis and cystic developmentSemantic proximity Read ↗

The molecular pathogenesis of HIV-1 associated nephropathy: recent advances.Semantic proximity Read ↗

Glomerulosclerosis and viral gene expression in HIV-transgenic mice: Role of nefSemantic proximity Read ↗

Tubular phenotypic change in progressive tubulointerstitial fibrosis in human glomerulonephritis.Semantic proximity Read ↗

Critical role for Nef in HIV-1-induced podocyte dedifferentiation.Semantic proximity Read ↗

The influence of tubular phenotypic changes on the development of diffuse interstitial fibrosis in renal allografts.Semantic proximity Read ↗

Nephropathy in human immunodeficiency virus-1 transgenic mice is due to renal transgene expression.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.