Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Association of common copy number variants at the glutathione S-transferase genes and rare novel genomic changes with schizophrenia.

Molecular psychiatry - 1 Oct 2010

Rodríguez-Santiago B, Brunet A, Sobrino B, Serra-Juhé C, Flores R, Armengol Ll, Vilella E, Gabau E, Guitart M, Guillamat R, Martorell L, Valero J, Gutiérrez-Zotes A, Labad A, Carracedo A, Estivill X, Pérez-Jurado L A

Abstract excerpt

Copy number variants (CNVs) are a substantial source of human genetic diversity, influencing the variable susceptibility to multifactorial disorders. Schizophrenia is a complex illness thought to be caused by a number of genetic and environmental effects, few of which have been clearly defined. Recent reports have found several low prevalent CNVs associated with the disease. We have used a multiplex...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Aged
Share to Gene Dosage

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Gene Duplication

Share to Genomics

Share to Glutathione Transferase

Share to Prevalence

Share to Risk Factors

Share to Schizophrenia

Source

Identifiers

PMID: 19528963
DOI: 10.1038/mp.2009.53

Genes

GSTM1
SSTR5

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Association of common copy number variants at the glutathione S-transferase genes and rare novel genomic changes with schizophrenia.PMID 19528963Read ↗

Associations of common copy number variants in glutathione S-transferase mu 1 and D-dopachrome tautomerase-like protein genes with risk of schizophrenia in a Japanese population.Semantic proximity Read ↗

Support for the involvement of large copy number variants in the pathogenesis of schizophrenia.Semantic proximity Read ↗

Copy Number Variations in Schizophrenia: Critical Review and New Perspectives on Concepts of Genetics and DiseaseSemantic proximity Read ↗

Rare chromosomal deletions and duplications increase risk of schizophrenia.Semantic proximity Read ↗

Comprehensive copy number variant (CNV) analysis of neuronal pathways genes in psychiatric disorders identifies rare variants within patients.Semantic proximity Read ↗

Detecting large copy number variants using exome genotyping arrays in a large Swedish schizophrenia sample.Semantic proximity Read ↗

A genome-wide investigation of SNPs and CNVs in schizophrenia.Semantic proximity Read ↗

A case-control study and meta-analysis of association between a common copy number variation of the glutathione S-transferase mu 1 (GSTM1) gene and schizophrenia.Semantic proximity Read ↗

Characterization of Single Gene Copy Number Variants in Schizophrenia.Semantic proximity Read ↗

Genomic copy number variations: A breakthrough in our knowledge on schizophrenia etiology?Semantic proximity Read ↗

CNV analysis in a large schizophrenia sample implicates deletions at 16p12.1 and SLC1A1 and duplications at 1p36.33 and CGNL1.Semantic proximity Read ↗

Large recurrent microdeletions associated with schizophrenia.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.