Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Meta-analysis of genome-wide linkage studies across autoimmune diseases.

European journal of human genetics : EJHG - 1 Feb 2009

Forabosco Paola, Bouzigon Emmanuelle, Ng Mandy Y, Hermanowski Jane, Fisher Sheila A, Criswell Lindsey A, Lewis Cathryn M

Abstract excerpt

Autoimmune diseases are chronic disorders initiated by a loss of immunologic tolerance to self-antigens. They cluster within families, and patients may be diagnosed with more than one disease, suggesting pleiotropic genes are involved in the aetiology of different diseases. To identify potential...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Autoimmune Diseases
Share to CTLA4 gene
Share to PTPN22 gene

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Genetic Predisposition to Disease

Share to Genome-Wide Association Study

Share to Major Histocompatibility Complex

Source

Open in PubMed Open full text

Identifiers

PMID: 18781189
DOI: 10.1038/ejhg.2008.163
PMCID: PMC2986058

Genes

CTLA4
PTPN22

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 2 Sept 2026. Prepared record publication-18781189-2026-09-02.2. Manifest publication-graph-cohort-2026-09-02.2.

Source receipt: PubMed via Helena Literature Corpus, retrieved 2 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Meta-analysis of genome-wide linkage studies across autoimmune diseases.PMID 18781189Read ↗

Systemic lupus erythematosus susceptibility loci defined by genome scan meta-analysis.Semantic proximity Read ↗

Multi-trait and cross-population genome-wide association studies across autoimmune and allergic diseases identify shared and distinct genetic componentSemantic proximity Read ↗

Pervasive sharing of genetic effects in autoimmune disease.Semantic proximity Read ↗

Meta-analysis of shared genetic architecture across ten pediatric autoimmune diseases.Semantic proximity Read ↗

Genetic mapping across autoimmune diseases reveals shared associations and mechanisms.Semantic proximity Read ↗

Genetic variation associated with cardiovascular risk in autoimmune diseases.Semantic proximity Read ↗

Joint analysis reveals shared autoimmune disease associations and identifies common mechanismsSemantic proximity Read ↗

PheWAS and cross-disorder analyses reveal genetic architecture, pleiotropic loci and phenotypic correlations across 11 autoimmune disordersSemantic proximity Read ↗

Emerging patterns of genetic overlap across autoimmune disordersSemantic proximity Read ↗

A comprehensive analysis of shared loci between systemic lupus erythematosus (SLE) and sixteen autoimmune diseases reveals limited genetic overlap.Semantic proximity Read ↗

HLA , CTLA-4 and PTPN22 : the shared genetic master-key to autoimmunity?Semantic proximity Read ↗

PheWAS and cross-disorder analysis reveal genetic architecture, pleiotropic loci and phenotypic correlations across 11 autoimmune disorders.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.