Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

The role of ace gene polymorphism in the development of angioedema secondary to angiotensin converting enzyme inhibitors and angiotensin II receptor blockers.

Allergologia et immunopathologia - 1 Jan 2000

Gulec M, Caliskaner Z, Tunca Y, Ozturk S, Bozoglu E, Gul D, Erel F, Kartal O, Karaayvaz M

Abstract excerpt

BACKGROUND: Angiotensin Converting Enzyme inhibitors (ACEi) may cause angioedema, with an incidence of 0.1 % to 1 %, which may be life-threatening. ACEi induce angioedema by increasing the levels of bradykinin. Angiotensin II receptor blockers (ATRB), have a pharmacological profile similar to ACE...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Aged
Share to Angioedema
Share to Angiotensin-Converting Enzyme Inhibitors

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Angiotensin II

Share to Angiotensin II Type 1 Receptor Blockers

Share to Genetic Predisposition to Disease

Share to Peptidyl-Dipeptidase A

Share to Polymorphism, Genetic

Source

Identifiers

PMID: 18680700

Genes

ACE

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 2 Sept 2026. Prepared record publication-18680700-2026-09-02.2. Manifest publication-graph-cohort-2026-09-02.2.

Source receipt: PubMed via Helena Literature Corpus, retrieved 2 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

The role of ace gene polymorphism in the development of angioedema secondary to angiotensin converting enzyme inhibitors and angiotensin II receptor blockers.PMID 18680700Read ↗

Genetic susceptibility to angiotensin-converting enzyme-inhibitor induced angioedema: A systematic review and evaluation of methodological approaches.Semantic proximity Read ↗

Potential genetic risk factors in angiotensin-converting enzyme-inhibitor-induced angio-oedema.Semantic proximity Read ↗

<i>ACE</i> PolymorphismsSemantic proximity Read ↗

Acute adverse reactions associated with angiotensin-converting enzyme inhibitors: genetic factors and therapeutic implications.Semantic proximity Read ↗

Pharmacogenetic markers of development of angioneurotic edema as a secondary side effect to enalapril in patients with essential arterial hypertension.Semantic proximity Read ↗

Association of Variants Near the Bradykinin Receptor B2 Gene With Angioedema in Patients Taking ACE Inhibitors.Semantic proximity Read ↗

Common variants near the bradykinin receptor B<sub>2</sub>gene are associated with angioedema induced by angiotensin-converting-enzyme inhibitor treatment - a genome wide association studySemantic proximity Read ↗

Association of insertion/deletion polymorphism of the angiotensin-converting enzyme gene with angio-oedema accompanying chronic urticaria but not chronic urticaria without angio-oedema or the autologous serum skin test response.Semantic proximity Read ↗

Identification of genetic factors associated with susceptibility to angiotensin-converting enzyme inhibitors-induced cough.Semantic proximity Read ↗

Angiotensin-Converting Enzyme I/D Polymorphism in Behçet’s DiseaseSemantic proximity Read ↗

Pharmacogenetic predictors of angiotensin-converting enzyme inhibitor-induced cough: the role of ACE, ABO, and BDKRB2 genes.Semantic proximity Read ↗

ACE inhibitor-mediated angioedemaSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.