Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Human MIP synthase splice variants in bipolar disorder.

Bipolar disorders - 1 Nov 2007

Shamir Alon, Shaltiel Galit, Mark Shirly, Bersudsky Yuly, Belmaker Robert H, Agam Galila

Abstract excerpt

OBJECTIVES: Alternative splicing allows the production of multiple gene products with different functions from a given sequence, affecting cellular function control. Tissue-specific splicing is most prevalent in the brain. We therefore investigate whether splice variants contribute to complex psychiatric disorders. A database search suggested that the myo-inositol-1-phosphate (MIP) synthase gene, possibly...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Alternative Splicing
Share to Bipolar Disorder
Share to Exons

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Frontal Lobe

Share to Gene Expression

Share to Genetic Variation

Share to Lymphocytes

Share to Reverse Transcriptase Polymerase Chain Reaction

Source

Identifiers

PMID: 17988368
DOI: 10.1111/j.1399-5618.2007.00440.x

Genes

MIP

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 11 closest

Human MIP synthase splice variants in bipolar disorder.PMID 17988368Read ↗

A human myo-inositol monophosphatase gene (IMPA2) localized in a putative susceptibility region for bipolar disorder on chromosome 18p11.2: genomic structure and polymorphism screening in manic-depressive patients.Semantic proximity Read ↗

Transcriptomic abnormalities in peripheral blood in bipolar disorder, and discrimination of the major psychosesSemantic proximity Read ↗

Transcriptome profiling as a biological marker for bipolar disorder sub-phenotypes.Semantic proximity Read ↗

Genomic structure and novel variants of myo-inositol monophosphatase 2 (IMPA2).Semantic proximity Read ↗

Alternative ion channel splicing in mesial temporal lobe epilepsy and Alzheimer's diseaseSemantic proximity Read ↗

A promoter haplotype of the inositol monophosphatase 2 gene (IMPA2) at 18p11.2 confers a possible risk for bipolar disorder by enhancing transcription.Semantic proximity Read ↗

In silico identification of transcripts and SNPs from a region of 4p linked with bipolar affective disorder.Semantic proximity Read ↗

Examination of IMPA1 and IMPA2 genes in manic-depressive patients: association between IMPA2 promoter polymorphisms and bipolar disorder.Semantic proximity Read ↗

Evidence for association of the myo-inositol monophosphatase 2 (IMPA2) gene with schizophrenia in Japanese samples.Semantic proximity Read ↗

Human 1-D-myo-inositol-3-phosphate synthase is functional in yeast.Semantic proximity Read ↗

Molecular Evidence for Mitochondrial Dysfunction in Bipolar DisorderSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.