Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Sensitivity of the hypothalamic paraventricular nucleus to the locomotor-activating effects of neuromedin U in obesity.

Brain research - 12 Sept 2007

Novak Colleen M, Zhang Minzhi, Levine James A

Abstract excerpt

Obesity is associated with a decrease in energy expenditure relative to energy intake. The decrease in physical activity associated with obesity in several species, including humans, contributes to decreased energy expenditure. Several hormones and neuropeptides that affect appetite also modulate physical activity, including neuromedin U (NMU), a peptide found in the gut and brain. We have demonstrated that NMU...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Dietary Fats
Share to Energy Metabolism
Share to Exploratory Behavior

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Motor Activity

Share to Neuropeptides

Share to Physical Conditioning, Animal

Share to Rats, Sprague-Dawley

Share to Thermogenesis

Source

Open in PubMed Open full text

Identifiers

PMID: 17706946
DOI: 10.1016/j.brainres.2007.06.055
PMCID: PMC2735201

Genes

NMU

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Sensitivity of the hypothalamic paraventricular nucleus to the locomotor-activating effects of neuromedin U in obesity.PMID 17706946Read ↗

Knockdown of Endogenous Nucb2/Nesfatin-1 in the PVN Leads to Obese-Like Phenotype and Abolishes the Metformin- and Stress-Induced Thermogenic Response in Rats.Semantic proximity Read ↗

Identification of a Thermogenic Target in the Dorsal Raphe Nucleus for Weight ManagementSemantic proximity Read ↗

Pre-existing differences in motivation for food and sensitivity to cocaine-induced locomotion in obesity-prone rats.Semantic proximity Read ↗

Discrete TrkB-expressing neurons of the dorsomedial hypothalamus regulate feeding and thermogenesisSemantic proximity Read ↗

Ventromedial nucleus neurons are less sensitive to leptin excitation in rats bred to develop diet-induced obesity.Semantic proximity Read ↗

betaAR signaling required for diet-induced thermogenesis and obesity resistance.Semantic proximity Read ↗

Region-specific differences in brain melanocortin receptors in rats of the lean phenotype.Semantic proximity Read ↗

Contribution of brown adipose tissue activity to the control of energy balance by GLP-1 receptor signalling in mice.Semantic proximity Read ↗

Effect of acute and chronic caloric restriction and metabolic glucoprivation on spontaneous physical activity in obesity-prone and obesity-resistant rats.Semantic proximity Read ↗

Alterations in activity and energy expenditure contribute to lean phenotype in Fischer 344 rats lacking the cholecystokinin-1 receptor gene.Semantic proximity Read ↗

Rhythmic neuronal activities of the rat nucleus of the solitary tract are impaired by short-term high fat diet - implications for daily control of satiety by the brainstemSemantic proximity Read ↗

Oxytocin signaling in the posterior hypothalamus prevents hyperphagic obesity in miceSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.