Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Amplification and Overexpression of CACNA1E Correlates with Relapse in Favorable Histology Wilms' Tumors

15 Dec 2006

Abstract excerpt

PURPOSE: The most well established molecular markers of poor outcome in Wilms' tumor are loss of heterozygosity at chromosomes 1p and/or 16q, although to date no specific genes at these loci have been identified. We have previously shown a link between genomic gain of chromosome 1q and tumor relapse and sought to further elucidate the role of genes on 1q in treatment failure. EXPERIMENTAL DESIGN: Microarray-based...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Renal and related cancers
Share to Tuberous Sclerosis Complex Research
Share to Wnt/β-catenin signaling in development and cancer

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Source

Identifiers

PMID: 17189400
DOI: 10.1158/1078-0432.ccr-06-1567

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Amplification and Overexpression of CACNA1E Correlates with Relapse in Favorable Histology Wilms' TumorsPMID 17189400Read ↗

Blastemal Expression of Type I Insulin-Like Growth Factor Receptor in Wilms' Tumors Is Driven by Increased Copy Number and Correlates with RelapseSemantic proximity Read ↗

Gain of 1q is associated with adverse outcome in favorable histology Wilms' tumors.Semantic proximity Read ↗

Genomic profiling by whole-genome single nucleotide polymorphism arrays in Wilms tumor and association with relapse.Semantic proximity Read ↗

Analysis by array CGH of genomic changes associated with the progression or relapse of Wilms' tumourSemantic proximity Read ↗

Chromosome 1q expression profiling and relapse in Wilms' tumourSemantic proximity Read ↗

Association of Chromosome 1q Gain With Inferior Survival in Favorable-Histology Wilms Tumor: A Report From the Children's Oncology Group.Semantic proximity Read ↗

Association between long interspersed nuclear element-1 methylation levels and relapse in Wilms tumorsSemantic proximity Read ↗

Genomic imbalances pinpoint potential oncogenes and tumor suppressors in Wilms tumorsSemantic proximity Read ↗

Cytogenetic abnormalities and clinical outcome in Wilms tumor: A study by the U.K. cancer cytogenetics group and the U.K. Children's Cancer Study Group†Semantic proximity Read ↗

Molecular characterization reveals subclasses of 1q gain in intermediate-risk Wilms tumorsSemantic proximity Read ↗

Subsets of Very Low Risk Wilms Tumor Show Distinctive Gene Expression, Histologic, and Clinical FeaturesSemantic proximity Read ↗

Decreased E-cadherin expression correlates with higher stage of Wilms' tumors.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.