Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Enhanced natural killer activity and production of pro-inflammatory cytokines in mice selected for high acute inflammatory response (AIRmax).

Immunology - 1 Mar 2007

Castoldi Lindsey, Golim Marjorie Assis, Filho Orlando Garcia Ribeiro, Romagnoli Graziela Gorete, Ibañez Olga Célia Martinez, Kaneno Ramon

Abstract excerpt

Strains of mice with maximal and minimal acute inflammatory responsiveness (AIRmax and AIRmin, respectively) were developed through selective breeding based on their high- or low-acute inflammatory responsiveness. Previous reports have shown that AIRmax mice are more resistant to the development of a variety of tumours than AIRmin mice, including spontaneous metastasis of murine melanoma. Natural killer activity...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Acute Disease
Share to Cytotoxicity, Immunologic

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Immunity, Innate

Share to Immunophenotyping

Share to Inflammation Mediators

Share to Killer Cells, Natural

Share to Spleen

Share to T-Lymphocyte Subsets

Source

Open in PubMed Open full text

Identifiers

PMID: 17163963
DOI: 10.1111/j.1365-2567.2006.02513.x
PMCID: PMC2265892

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 6 Sept 2026. Manifest publication-graph-cohort-2026-09-06.1.

Source receipt: pubmed, retrieved 6 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Enhanced natural killer activity and production of pro-inflammatory cytokines in mice selected for high acute inflammatory response (AIRmax).PMID 17163963Read ↗

Resistance to melanoma metastases in mice selected for high acute inflammatory response.Semantic proximity Read ↗

A new model of outbred genetically selected mice which present a strong acute inflammatory response in the absence of complement component C5.Semantic proximity Read ↗

Effect of genetic modification of acute inflammatory responsiveness on tumorigenesis in the mouseSemantic proximity Read ↗

Genetic selection for high acute inflammatory response confers resistance to lung carcinogenesis in the mouse.Semantic proximity Read ↗

Slc11a1 (formerly NRAMP1) gene modulates both acute inflammatory reactions and pristane-induced arthritis in mice.Semantic proximity Read ↗

Innate resistance to infection by intracellular bacterial pathogens differs in mice selected for maximal or minimal acute inflammatory response.Semantic proximity Read ↗

Distinct early inflammatory events during ear tissue regeneration in mice selected for high inflammation bearing Slc11a1 R and S alleles.Semantic proximity Read ↗

Inverse genetic predisposition to colon versus lung carcinogenesis in mouse lines selected based on acute inflammatory responsiveness.Semantic proximity Read ↗

Genetic determinants of acute inflammation regulate Salmonella infection and modulate Slc11a1 gene (formerly Nramp1) effects in selected mouse lines.Semantic proximity Read ↗

Specific dysregulation of IFNγ production by natural killer cells confers susceptibility to viral infection.Semantic proximity Read ↗

Aryl hydrocarbon receptor polymorphism modulates DMBA-induced inflammation and carcinogenesis in phenotypically selected mice.Semantic proximity Read ↗

Kit (W-sh) mice develop earlier and more severe experimental autoimmune encephalomyelitis due to absence of immune suppression.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.