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Article

The C3b/C4b receptor is recognized by the Knops, McCoy, Swain-langley, and York blood group antisera.

The Journal of experimental medicine - 1 May 1991

Moulds J M, Nickells M W, Moulds J J, Brown M C, Atkinson J P

Abstract excerpt

Erythrocytes (E) lacking high incidence blood group antigens were screened by an antiglobulin test with a monoclonal antibody to human complement receptor type 1 (CR1; C3b/C4b receptor; CD35). Some examples of E lacking Knops, McCoy, Swain-Langley, and York antigens, a serologically related group...

Topics

Alleles
Antibodies, Monoclonal
Blood Group Antigens
Carrier Proteins
Complement Inactivator Proteins
Epitopes
Erythrocytes
Glycoproteins
Hemagglutination
Humans
Immune Sera
Isoantigens

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Phenotype

Polymorphism, Genetic

Precipitin Tests

Receptors, Complement

Receptors, Complement 3b

Source

Open in PubMed Open full text

Identifiers

PMID: 1708809
DOI: 10.1084/jem.173.5.1159
PMCID: PMC2118866

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 30 Aug 2026. Prepared record publication-1708809-2026-08-30.1. Manifest publication-graph-cohort-2026-08-30.1.

Source receipt: PubMed via Helena Literature Corpus, retrieved 30 Aug 2026.

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The C3b/C4b receptor is recognized by the Knops, McCoy, Swain-langley, and York blood group antisera.PMID 1708809Read ↗

Antiglobulin testing for CR1-related (Knops/McCoy/Swain-Langley/York) blood group antigens: negative and weak reactions are caused by variable expression of CR1.Semantic proximity Read ↗

Analysis of complement receptor type 1 expression on red blood cells in negative phenotypes of the Knops blood group system, according to CR1 gene allotype polymorphisms.Semantic proximity Read ↗

Molecular analysis of the York antigen of the Knops blood group system.Semantic proximity Read ↗

Complement receptor 1 red cell expression is not controlled by the In(Lu) gene.Semantic proximity Read ↗

Molecular identification of Knops blood group polymorphisms found in long homologous region D of complement receptor 1Semantic proximity Read ↗

Lack of evidence from studies of soluble protein fragments that Knops blood group polymorphisms in complement receptor-type 1 are driven by malaria.Semantic proximity Read ↗

Expansion of the Knops blood group system and subdivision of Sl(a).Semantic proximity Read ↗

No Evidence that Knops Blood Group Polymorphisms Affect Complement Receptor 1 Clustering on Erythrocytes.Semantic proximity Read ↗

CR1 Knops blood group alleles are not associated with severe malaria in the Gambia.Semantic proximity Read ↗

Identification of the Kna/Knb polymorphism and a method for Knops genotyping.Semantic proximity Read ↗

A CR1 polymorphism associated with constitutive erythrocyte CR1 levels affects binding to C4b but not C3b.Semantic proximity Read ↗

Independence of Wright from many other blood group systems.Semantic proximity Read ↗

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