Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Behavioral motor dysfunction in Kv3-type potassium channel-deficient mice.

Genes, brain, and behavior - 1 Aug 2006

Joho R H, Street C, Matsushita S, Knöpfel T

Abstract excerpt

The voltage-gated potassium channels Kv3.1 and Kv3.3 are expressed in several distinct neuronal subpopulations in brain areas known to be involved in motor control such as cortex, basal ganglia and cerebellum. Depending on the lack of Kv3.1 or Kv3.3 channel subunits, mutant mice show different Kv...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cerebellum
Share to Gait Disorders, Neurologic
Share to Learning

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Learning Disabilities

Share to Mice, Inbred ICR

Share to Movement

Share to Movement Disorders

Share to Synaptic Transmission

Source

Identifiers

PMID: 16923152
DOI: 10.1111/j.1601-183X.2005.00184.x

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 2 Sept 2026. Prepared record publication-16923152-2026-09-02.2. Manifest publication-graph-cohort-2026-09-02.2.

Source receipt: PubMed via Helena Literature Corpus, retrieved 2 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Behavioral motor dysfunction in Kv3-type potassium channel-deficient mice.PMID 16923152Read ↗

Alcohol hypersensitivity, increased locomotion, and spontaneous myoclonus in mice lacking the potassium channels Kv3.1 and Kv3.3.Semantic proximity Read ↗

Purkinje-cell-restricted restoration of Kv3.3 function restores complex spikes and rescues motor coordination in Kcnc3 mutants.Semantic proximity Read ↗

Allele-dependent changes of olivocerebellar circuit properties in the absence of the voltage-gated potassium channels Kv3.1 and Kv3.3.Semantic proximity Read ↗

Rescue of motor coordination by Purkinje cell-targeted restoration of Kv3.3 channels in Kcnc3-null mice requires Kcnc1.Semantic proximity Read ↗

Cognitive deficits caused by a disease-mutation in the α3 Na(+)/K(+)-ATPase isoform.Semantic proximity Read ↗

Neurological abnormalities in caveolin-1 knock out mice.Semantic proximity Read ↗

Phenotype Distinctions in Mice Deficient in the Neuron-Specific α3 Subunit of Na,K-ATPase: Atp1a3tm1Ling/+ and Atp1a3 +/D801Y.Semantic proximity Read ↗

Cerebellar purkinje cell dysfunction contributes to gait impairments in Shank3-mutant mice.Semantic proximity Read ↗

Characterization of cognitive deficits in mice with an alternating hemiplegia-linked mutation.Semantic proximity Read ↗

The puzzling role of TRPC3 channels in motor coordination.Semantic proximity Read ↗

Motor deficits in homozygous and heterozygous p/q-type calcium channel mutants.Semantic proximity Read ↗

Spinocerebellar ataxia type 13 mutant potassium channel alters neuronal excitability and causes locomotor deficits in zebrafish.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.