Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Glomerular expression of neuronal activity-regulated pentraxin precedes the development of anti-Thy-1-induced progressive glomerulosclerosis.

Kidney international - 1 Oct 2006

Aben J A, Ijpelaar D H, Baelde H, Worley P, Noble N, Bruijn J A, de Heer E

Abstract excerpt

Although it is clear that genetic predispositions play a role in progressive glomerulosclerosis, identification of specific genes is difficult because of natural genetic heterogeneity among individuals. We have reported a differential susceptibility to progressive glomerulosclerosis after induction of experimental glomerulonephritis anti-Thy-1 nephritis in Lewis rat substrains. Glomerular lesions in...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to C-Reactive Protein
Share to Glomerulonephritis

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Isoantibodies

Share to Kidney Glomerulus

Share to Nephrectomy

Share to Nephritis

Share to Podocytes

Share to Rats, Inbred Lew

Source

Identifiers

PMID: 16900090
DOI: 10.1038/sj.ki.5001757

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Glomerular expression of neuronal activity-regulated pentraxin precedes the development of anti-Thy-1-induced progressive glomerulosclerosis.PMID 16900090Read ↗

Genes expressed by the kidney, but not by bone marrow-derived cells, underlie the genetic predisposition to progressive glomerulosclerosis after mesangial injury.Semantic proximity Read ↗

Genetic predisposition for glomerulonephritis-induced glomerulosclerosis in rats is linked to chromosome 1.Semantic proximity Read ↗

Global polysome analysis of normal and injured podocytesSemantic proximity Read ↗

Strain differences and the genetic basis of experimental autoimmune anti-glomerular basement membrane glomerulonephritis.Semantic proximity Read ↗

cDNA microarray analysis of adaptive changes after renal ablation in a sclerosis-resistant mouse strain.Semantic proximity Read ↗

Proliferative autoimmune glomerulonephritis in rats: a model for autoimmune glomerulonephritis in humans.Semantic proximity Read ↗

Genetic loci modulate macrophage activity and glomerular damage in experimental glomerulonephritis.Semantic proximity Read ↗

Genetic susceptibility to experimental autoimmune glomerulonephritis in the Wistar Kyoto rat.Semantic proximity Read ↗

Genetic study of gold-salt-induced immune disorders in the rat.Semantic proximity Read ↗

Mercury-induced renal autoimmunity: changes in RT6+ T-lymphocytes of susceptible and resistant rats.Semantic proximity Read ↗

Strain susceptibility to active induction and passive transfer of experimental autoimmune glomerulonephritis in the rat.Semantic proximity Read ↗

The inheritance of glomerulosclerosis in mice is controlled by multiple quantitative trait loci.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.