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Article

Compensation of endothelium-dependent responses in coronary circulation of eNOS-deficient mice.

Journal of cardiovascular pharmacology - 1 Jul 2005

Chlopicki Stefan, Kozlovski Valery I, Lorkowska Barbara, Drelicharz Lukasz, Gebska Anna

Abstract excerpt

Nitric oxide plays a fundamental role in the regulation of blood flow. Here we analyzed compensatory mechanisms for the genetic eNOS deficiency in aorta and in coronary circulation. Vasodilation induced by acetylcholine, bradykinin, adenosine, and ADP as well as by S-nitroso-penicillamine (SNAP) was assessed in isolated aorta and in isolated mouse hearts from eNOS-/- and age-matched eNOS+/+ mice. In aorta from...

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Source

Identifiers

PMID: 15965363
DOI: 10.1097/01.fjc.0000164093.88821.00

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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