Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

APOA5 gene variants, lipoprotein particle distribution, and progression of coronary heart disease: results from the LOCAT study.

Journal of lipid research - 1 Apr 2004

Talmud Philippa J, Martin Steve, Taskinen Marja-Riitta, Frick M Heikki, Nieminen Markku S, Kesäniemi Y Antero, Pasternack Amos, Humphries Steve E, Syvänne Mikko

Abstract excerpt

Animal and human studies support a role for apolipoprotein A-V (apoA-V) in triglyceride (TG) metabolism. We examined the relationship of APOA5 -1131T>C and S19W with lipid subfractions and progression of atherosclerosis in the Lopid Coronary Angiography Trial. Compared with -1131TT men (n = 242),...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Apolipoprotein A-V
Share to Apolipoproteins
Share to Apolipoproteins A

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Arteriosclerosis

Share to Coronary Disease

Share to Lipoproteins

Share to Lipoproteins, VLDL

Share to Particle Size

Source

Identifiers

PMID: 14729863
DOI: 10.1194/jlr.M300458-JLR200

Genes

APOA5

Variants

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 1 Sept 2026. Prepared record publication-14729863-2026-09-01.1. Manifest publication-graph-cohort-2026-09-01.1.

Source receipt: PubMed via Helena Literature Corpus, retrieved 1 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

APOA5 gene variants, lipoprotein particle distribution, and progression of coronary heart disease: results from the LOCAT study.PMID 14729863Read ↗

The -1131 T>C and S19W APOA5 gene polymorphisms are associated with high levels of triglycerides and apolipoprotein C-III, but not with coronary artery disease: an angiographic study.Semantic proximity Read ↗

Influence of the APOA5 locus on plasma triglyceride, lipoprotein subclasses, and CVD risk in the Framingham Heart Study.Semantic proximity Read ↗

Triglyceride-mediated pathways and coronary disease: collaborative analysis of 101 studies.Semantic proximity Read ↗

APOA5 variants predispose hyperlipidemic patients to atherogenic dyslipidemia and subclinical atherosclerosis.Semantic proximity Read ↗

Variants at the APOA5 locus, association with carotid atherosclerosis, and modification by obesity: the Framingham Study.Semantic proximity Read ↗

APOA5 genetic variants are markers for classic hyperlipoproteinemia phenotypes and hypertriglyceridemia.Semantic proximity Read ↗

Relative contribution of variation within the APOC3/A4/A5 gene cluster in determining plasma triglycerides.Semantic proximity Read ↗

Apolipoprotein A-V: a potential modulator of plasma triglyceride levels in Turks.Semantic proximity Read ↗

Candidate genetic analysis of plasma high-density lipoprotein-cholesterol and severity of coronary atherosclerosis.Semantic proximity Read ↗

The apolipoprotein A5 -1131T>C promoter polymorphism in Koreans: association with plasma APOA5 and serum triglyceride concentrations, LDL particle size and coronary artery disease.Semantic proximity Read ↗

APOA5 gene polymorphism modulates levels of triglyceride, HDL cholesterol and FERHDL but is not a risk factor for coronary artery disease.Semantic proximity Read ↗

Haplotype analyses of the APOA5 gene in patients with familial combined hyperlipidemia.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.