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Article

p53 mutations in bladder cancer: evidence for exogenous versus endogenous risk factors.

Cancer research - 1 Nov 2003

Schroeder Jane C, Conway Kathleen, Li Yu, Mistry Kusum, Bell Douglas A, Taylor Jack A

Abstract excerpt

Bladder cancer is associated with smoking, occupational exposures, and glutathione S-transferase (GST) M1 and N-acetyltransferase (NAT) 2 polymorphisms that may influence carcinogen metabolism, but somatic p53mutations are often CpG dinucleotide G:C-A:T transitions that can occur spontaneously. We conducted a case-control study to determine whether p53mutation characteristics might distinguish cases with...

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Source

Identifiers

PMID: 14612556

Genes

GSTM1
NAT2
NAT1

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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p53 mutations in bladder cancer: evidence for exogenous versus endogenous risk factors.PMID 14612556Read ↗

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