Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Promoter but not exon 7 polymorphism of endothelial nitric oxide synthase affects training-induced correction of endothelial dysfunction.

Arteriosclerosis, thrombosis, and vascular biology - 1 Oct 2003

Erbs Sandra, Baither Yves, Linke Axel, Adams Volker, Shu Yanwen, Lenk Karsten, Gielen Stephan, Dilz Rainer, Schuler Gerhard, Hambrecht Rainer

Abstract excerpt

UNLABELLED: Background- Polymorphisms in the promoter (T-786C) and exon 7 (G894T) of the endothelial nitric oxide synthase (eNOS) gene were shown to be associated with reduced vascular NO production or increased proteolytic cleavage of eNOS. Therefore, we aimed to determine the effects of these p...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Coronary Disease
Share to DNA
Share to Endothelium, Vascular

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Exercise

Share to Nitric Oxide Synthase

Share to Promoter Regions, Genetic

Share to Vasodilation

Source

Identifiers

PMID: 12907461
DOI: 10.1161/01.ATV.0000090128.11465.18

Genes

CAD

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 1 Sept 2026. Prepared record publication-12907461-2026-09-01.1. Manifest publication-graph-cohort-2026-09-01.1.

Source receipt: PubMed via Helena Literature Corpus, retrieved 1 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Promoter but not exon 7 polymorphism of endothelial nitric oxide synthase affects training-induced correction of endothelial dysfunction.PMID 12907461Read ↗

Both T-786C and G894T polymorphism of endothelial nitric oxide synthase affect in-vitro endothelium-dependent relaxation of internal mammary artery rings from patients with coronary artery disease.Semantic proximity Read ↗

A promoter polymorphism of the endothelial nitric oxide synthase gene is associated with reduced mRNA and protein expression in failing human myocardium.Semantic proximity Read ↗

The T-786C endothelial nitric oxide synthase genotype is a novel risk factor for coronary artery disease in Caucasian patients of the GENICA study.Semantic proximity Read ↗

The T-786C and Glu298Asp polymorphisms of the endothelial nitric oxide gene affect the forearm blood flow responses of Caucasian hypertensive patients.Semantic proximity Read ↗

Exercise training improves muscle vasodilatation in individuals with T786C polymorphism of endothelial nitric oxide synthase gene.Semantic proximity Read ↗

The T(-786)C endothelial nitric oxide synthase genotype predicts cardiovascular mortality in high-risk patients.Semantic proximity Read ↗

The endothelial nitric oxide synthase -786 T>C polymorphism and the exercise-induced blood pressure and nitric oxide responses among men with elevated blood pressure.Semantic proximity Read ↗

Influence of eNOS gene polymorphism on cardiometabolic parameters in response to physical training in postmenopausal women.Semantic proximity Read ↗

Effect of exercise training on the cardiovascular and biochemical parameters in women with eNOS gene polymorphism.Semantic proximity Read ↗

Women with TT genotype for eNOS gene are more responsive in lowering blood pressure in response to exercise.Semantic proximity Read ↗

Allele-specific regulation of matrix metalloproteinase-7 promoter activity is associated with coronary artery luminal dimensions among hypercholesterolemic patients.Semantic proximity Read ↗

Effects of endothelial nitric oxide synthase gene polymorphisms on platelet function, nitric oxide release, and interactions with estradiol.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.