Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Targets of oxidative stress in yeast sod mutants.

Biochimica et biophysica acta - 17 Mar 2003

Pereira M D, Herdeiro R S, Fernandes P N, Eleutherio E C A, Panek A D

Abstract excerpt

Eukaryotic cells have developed mechanisms to rapidly respond towards the environment by changing the expression of a series of genes. There is increasing evidence that reactive oxygen species (ROS), besides causing damage, may also fulfill an important role as second messengers involved in signal transduction. Recently, we have demonstrated that deletion of SOD1 is beneficial for the acquisition of tolerance...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Gene Expression Regulation, Fungal
Share to Glutathione
Share to Metallothionein

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Oxidation-Reduction

Share to Second Messenger Systems

Share to Superoxide Dismutase

Share to Superoxide Dismutase-1

Share to Vitamin K 3

Source

Identifiers

PMID: 12595095
DOI: 10.1016/s0304-4165(03)00003-5

Genes

SOD1

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Targets of oxidative stress in yeast sod mutants.PMID 12595095Read ↗

Diauxic shift-induced stress resistance against hydroperoxides in Saccharomyces cerevisiae is not an adaptive stress response and does not depend on functional mitochondria.Semantic proximity Read ↗

Overexpression of human SOD1 in VDAC1-less yeast restores mitochondrial functionality modulating beta-barrel outer membrane protein genes.Semantic proximity Read ↗

Yeast glutathione reductase is required for protection against oxidative stress and is a target gene for yAP-1 transcriptional regulation.Semantic proximity Read ↗

Requirement of glutathione for Sod1 activation during lifespan extension.Semantic proximity Read ↗

Growth on ethanol results in co-ordinated Saccharomyces cerevisiae response to inactivation of genes encoding superoxide dismutases.Semantic proximity Read ↗

Adaptative response to enhanced basal oxidative damage in sod mutants from Saccharomyces cerevisiae.Semantic proximity Read ↗

Menadione stress in Saccharomyces cerevisiae strains deficient in the glutathione transferases.Semantic proximity Read ↗

Glutathione is an essential metabolite required for resistance to oxidative stress in the yeast Saccharomyces cerevisiae.Semantic proximity Read ↗

Cellular factors required for protection from hyperoxia toxicity in Saccharomyces cerevisiae.Semantic proximity Read ↗

Superoxide radicals have a protective role during H2O2 stress.Semantic proximity Read ↗

Glutathione, but not transcription factor Yap1, is required for carbon source-dependent resistance to oxidative stress in Saccharomyces cerevisiae.Semantic proximity Read ↗

Superoxide dismutase 1 acts as a nuclear transcription factor to regulate oxidative stress resistance.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.