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Article

The Lack of Thrombospondin-1 (TSP1) Dictates the Course of Wound Healing in Double-TSP1/TSP2-Null Mice

1 Sept 2002

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 12213711
DOI: 10.1016/s0002-9440(10)64243-5

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Related research

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The Lack of Thrombospondin-1 (TSP1) Dictates the Course of Wound Healing in Double-TSP1/TSP2-Null MicePMID 12213711Read ↗

Compound deletion of thrombospondin-1 and -2 results in a skeletal phenotype not predicted by the single gene knockouts.Semantic proximity Read ↗

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Global deletion of thrombospondin-1 increases cardiac and skeletal muscle capillarity and exercise capacity in mice.Semantic proximity Read ↗

Endogenous thrombospondin 1 protects the pressure-overloaded myocardium by modulating fibroblast phenotype and matrix metabolism.Semantic proximity Read ↗

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Thrombospondins: structure and regulation of expressionSemantic proximity Read ↗

Accelerated Wound Repair in ADAM-9 Knockout AnimalsSemantic proximity Read ↗

Thrombospondin-2 deficiency in growing mice alters bone collagen ultrastructure and leads to a brittle bone phenotype.Semantic proximity Read ↗

Thrombospondin-2 overexpression in the skin of transgenic mice reduces the susceptibility to chemically induced multistep skin carcinogenesis.Semantic proximity Read ↗

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