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Article

Two NOD Idd-associated intervals contribute synergistically to the development of autoimmune exocrinopathy (Sjögren's syndrome) on a healthy murine background.

Arthritis and rheumatism - 1 May 2002

Cha Seunghee, Nagashima Hiroyuki, Brown Vinette B, Peck Ammon B, Humphreys-Beher Michael G

Abstract excerpt

OBJECTIVE: The NOD mouse is genetically predisposed to the development of at least 2 autoimmune diseases, autoimmune diabetes and autoimmune exocrinopathy (AEC). More than 19 chromosomal intervals (referred to as Idd regions) that contribute to diabetes susceptibility in the NOD mouse model have been identified, but only 2 chromosomal intervals (associated with Idd3 and Idd5) have been shown to control...

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Source

Identifiers

PMID: 12115247
DOI: 10.1002/art.10258

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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