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Article

p57(KIP2) expression in normal islet cells and in hyperinsulinism of infancy.

Diabetes - 1 Dec 2001

Kassem S A, Ariel I, Thornton P S, Hussain K, Smith V, Lindley K J, Aynsley-Green A, Glaser B

Abstract excerpt

Most cases of hyperinsulinism of infancy (HI) are caused by mutations in either the sulfonylurea receptor-1 (SUR1) or the inward rectifying K(+) channel Kir6.2, two subunits of the beta-cell ATP-sensitive K(+) channel (K(ATP) channel). Histologically, HI can be divided into two major subtypes. The diffuse form is recessively inherited and involves all beta-cells within the pancreas. Focal HI consists of...

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

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Source

Identifiers

PMID: 11723059
DOI: 10.2337/diabetes.50.12.2763

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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p57(KIP2) expression in normal islet cells and in hyperinsulinism of infancy.PMID 11723059Read ↗

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