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Article

Two AraC/XylS family members can independently counteract the effect of repressing sequences upstream of the hilA promoter.

Molecular microbiology - 1 May 1999

Schechter L M, Damrauer S M, Lee C A

Abstract excerpt

During infection of its hosts, Salmonella enterica serovar Typhimurium (S. typhimurium) enters the epithelial cells of the small intestine. This process requires a number of invasion genes encoded on Salmonella pathogenicity island 1 (SPI1), a 40 kb stretch of DNA located near minute 63 of the S. typhimurium chromosome. Expression of S. typhimurium SPI1 invasion genes is activated by the transcription factor...

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Share to Gene Expression Regulation, Bacterial
Share to Regulatory Sequences, Nucleic Acid

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Repressor Proteins

Share to Salmonella typhimurium

Share to Sequence Deletion

Share to Sequence Homology, Amino Acid

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Source

Identifiers

PMID: 10320584
DOI: 10.1046/j.1365-2958.1999.01381.x

Genes

SPI1

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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Two AraC/XylS family members can independently counteract the effect of repressing sequences upstream of the hilA promoter.PMID 10320584Read ↗

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