Article

Chronic exposure to low-concentration urban PM2.5 accelerates maladaptive repair after ischemic injury via mitochondrial dysfunction and lysosomal stress

2026-03-14

Abstract excerpt

<h4>Background</h4> Fine particulate matter (PM2.5), airborne particles with an aerodynamic diameter ≤2.5 μm that can penetrate deep into the lungs and enter the circulation, is increasingly recognized as a risk factor for chronic kidney disease (CKD) with long-term exposure. We previously demonstrated that high-dose PM2.5 exposure prior to ischemia–reperfusion injury (IRI) aggravates acute kidney injury (AKI). H...

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Literature Corpus work: debd24f8-b2f6-593f-9fa6-2fcd27b76fa4
DOI: 10.64898/2026.03.11.711056

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Chronic exposure to low-concentration urban PM2.5 accelerates maladaptive repair after ischemic injury via mitochondrial dysfunction and lysosomal stressDOI 10.64898/2026.03.11.711056Read

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