Back to search

Article

ANTXR1 Facilitates Tumor Growth in Glioma via Deactivating MAPK Signaling Pathway

2020-12-16

Abstract excerpt

<h4>Background: </h4> Gliomas are commonly known as primary brain tumors and associated with frequent recurrence and an unsatisfactory prognosis in despite of extensive research in the underlying molecular mechanisms. In this study, we aimed to examine the role of ANTXR1 in glioma tumorigenesis and explore its downstream regulatory mechanism. <h4>Methods:</h4> We detected overexpression of ANTXR1 in glioma cell li...

Topics

Open a Topic to create a Post that cites this publication.

Identifiers and source

Literature Corpus work
a16f9fe3-e60b-562c-9b99-78ccf2ea224a
DOI
10.21203/rs.3.rs-126784/v1
Open publication

Related research

Semantic proximity does not establish scientific evidence.

Click a neighbor to travelStep 1 · 12 closest
Interactive article relationship graphSelect a related publication card to move it into the centre and load its closest explainable connections. Solid lines are source-backed structured connections. Dashed lines are semantic discovery signals and are not scientific evidence.
ANTXR1&nbsp;Facilitates Tumor Growth in Glioma via Deactivating MAPK Signaling PathwayDOI 10.21203/rs.3.rs-126784/v1
Gliomas: Genetic alterations, mechanisms of metastasis, recurrence, drug resistance, and recent trends in molecular therapeutic options.Semantic proximity
The Role of ATRX in Glioma BiologySemantic proximity
Astrocyte elevated gene 1: biological functions and molecular mechanism in cancer and beyondSemantic proximity
Navigating Glioma Complexity: The Role of Abnormal Signaling Pathways in Shaping Future TherapiesSemantic proximity
GOPC:ROS1 and other ROS1 fusions represent a rare but recurrent drug target in a variety of glioma typesSemantic proximity
Decreased expression of NDRG1 in glioma is related to tumor progression and survival of patientsSemantic proximity
Interaction with AEG-1 and MDM2 is associated with glioma development and progression and correlates with poor prognosisSemantic proximity
Novel KDM1A inhibitors induce differentiation and apoptosis of glioma stem cells via unfolded protein response pathwaySemantic proximity
Novel mutations of SRPX facilitate the stemness and malignant progression of glioma.Semantic proximity
PSMC2/E2F1 Axis Promotes the Development and Progression of GliomaSemantic proximity
Necroptotic Signaling Orchestrates GBM Malignancy and Potentiates Temozolomide ResponseSemantic proximity
Astrocyte Elevated Gene-1 Interacts with Akt Isoform 2 to Control Glioma Growth, Survival, and PathogenesisSemantic proximity
Select a neighboring publication to make it the new centre.