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Selective silencing of the hypoxia-inducible factor 1 target gene BNIP3 by histone deacetylation and methylation in colorectal cancerDOI 10.1038/sj.onc.1209761
Aberrant Methylation and Silencing of the<i>BNIP3</i>Gene in Colorectal and Gastric CancerSemantic proximity
The role of the hypoxia-inducible BH3-only proteins BNIP3 and BNIP3L in cancerSemantic proximity
Aberrant DNA methylation associated with silencing BNIP3 gene expression in haematopoietic tumoursSemantic proximity
SET1B facilitates activation of the hypoxia response through site-specific histone methylationSemantic proximity
Hypoxia induces the expression of the pro-apoptotic gene BNIP3Semantic proximity
Chemoresistance of colorectal cancer to 5-fluorouracil is associated with silencing of the BNIP3 gene through aberrant methylationSemantic proximity
BNIP3 Expression Is Linked with Hypoxia-Regulated Protein Expression and with Poor Prognosis in Non–Small Cell Lung CancerSemantic proximity
Regulation of BNIP3 in Normal and Cancer CellsSemantic proximity
NUSAP1 gene silencing inhibits cell proliferation, migration and invasion through inhibiting DNMT1 gene expression in human colorectal cancerSemantic proximity
BNIP3 plays a role in hypoxic cell death in human epithelial cells that is inhibited by growth factors EGF and IGF.Semantic proximity
Poorer outcome in stromal HIF-2 alpha- and CA9-positive colorectal adenocarcinomas is associated with wild-type TP53 but not with BNIP3 promoter hypermethylation or apoptosis.Semantic proximity
<i>BNIP3</i> Is an RB/E2F Target Gene Required for Hypoxia-Induced AutophagySemantic proximity
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