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Article

The role of the hypoxia-inducible BH3-only proteins BNIP3 and BNIP3L in cancer

4 Sept 2007

Abstract excerpt

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Share to Autophagy in Disease and Therapy
Share to Cancer, Hypoxia, and Metabolism
Share to Heme Oxygenase-1 and Carbon Monoxide

Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 17805942
DOI: 10.1007/s10555-007-9080-0

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 8 Sept 2026.

Related research

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Research trail→

Click a neighbor to travelStep 1 · 12 closest

The role of the hypoxia-inducible BH3-only proteins BNIP3 and BNIP3L in cancerPMID 17805942Read ↗

Hypoxia induces the expression of the pro-apoptotic gene BNIP3Semantic proximity Read ↗

Regulation of BNIP3 in Normal and Cancer CellsSemantic proximity Read ↗

BNIP3 in Lung Cancer: To Kill or Rescue?Semantic proximity Read ↗

BNIP3 plays a role in hypoxic cell death in human epithelial cells that is inhibited by growth factors EGF and IGF.Semantic proximity Read ↗

Hypoxia-Induced Autophagy Is Mediated through Hypoxia-Inducible Factor Induction of BNIP3 and BNIP3L via Their BH3 DomainsSemantic proximity Read ↗

Roles of HIF1α- and HIF2α-regulated BNIP3 in hypoxia-induced injury of neuronsSemantic proximity Read ↗

Bcl-2 interacting protein 3 (BNIP3) promotes tumor growth in breast cancer under hypoxic conditions through an autophagy-dependent pathwaySemantic proximity Read ↗

BNIP3 Expression Is Linked with Hypoxia-Regulated Protein Expression and with Poor Prognosis in Non–Small Cell Lung CancerSemantic proximity Read ↗

Regulation of Mitochondrial Integrity, Autophagy and Cell Survival by BNIP3Semantic proximity Read ↗

BNIP3 subfamily BH3-only proteins: mitochondrial stress sensors in normal and pathological functionsSemantic proximity Read ↗

Selective silencing of the hypoxia-inducible factor 1 target gene BNIP3 by histone deacetylation and methylation in colorectal cancerSemantic proximity Read ↗

The HIF1α-inducible pro-cell death gene BNIP3 is a novel target of SIM2s repression through cross-talk on the hypoxia response elementSemantic proximity Read ↗

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