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Prefrontal parvalbumin neurons as a target for enhancing cognition in non-pathological and 22q11.2 microdeletion syndrome mice

2025-04-01

Abstract excerpt

A failure of organized communication in the PFC is thought to contribute to the emergence of cognitive impairments in psychiatric diseases, with attentional deficits occurring as a fundamental symptom across various conditions. The 22q11.2 microdeletion syndrome is a rare genetic condition that confers a high risk for developing psychiatric and neurodevelopmental disorders, and mouse models have been shown to disp...

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Literature Corpus work
1d59d487-437d-5291-a6ed-8bb54454e8de
DOI
10.1101/2025.03.31.645999
Open publication

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Prefrontal parvalbumin neurons as a target for enhancing cognition in non-pathological and 22q11.2 microdeletion syndrome miceDOI 10.1101/2025.03.31.645999
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