Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

GENETIC COMPONENT IN THE INFLAMMATORY RESPONSE INDUCED BY BACTERIAL LIPOPOLYSACCHARIDE

1 Nov 1998

Abstract excerpt

Multiple organ dysfunction syndrome (MODS) appears to be the result of a complex program influenced by multiple factors, including environmental, physiological, and immunological conditions. Thus, an uncontrolled inflammatory response following a stochastic event, the initial injury, is believed to be the cause for the development of this syndrome. Several lines of evidence suggest that a genetic component could...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Drug-Induced Hepatotoxicity and Protection
Share to Immune Response and Inflammation
Share to Sepsis Diagnosis and Treatment

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Source

Identifiers

PMID: 9840645
DOI: 10.1097/00024382-199811000-00002

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

GENETIC COMPONENT IN THE INFLAMMATORY RESPONSE INDUCED BY BACTERIAL LIPOPOLYSACCHARIDEPMID 9840645Read ↗

Genetic determinants influencing the response to injury, inflammation, and sepsis.Semantic proximity Read ↗

Novel Regulators of the Systemic Response to LipopolysaccharideSemantic proximity Read ↗

Identification of rat quantitative trait loci that regulate LPS-induced pro-inflammatory cytokine responses.Semantic proximity Read ↗

Genetic control of the innate immune response.Semantic proximity Read ↗

Identification of novel genes that mediate innate immunity using inbred mice.Semantic proximity Read ↗

Comparison of acute endotoxin-induced lesions in A/J and C57BL/6J mice.Semantic proximity Read ↗

The genetics of innate immunity.Semantic proximity Read ↗

Differential Expression of Inflammatory Cytokines and Stress Genes in Male and Female Mice in Response to a Lipopolysaccharide ChallengeSemantic proximity Read ↗

Mouse genetic background influences severity of immune responses following trauma-hemorrhage.Semantic proximity Read ↗

Comparison of the Susceptibilities of C57BL/6 and A/J Mouse Strains toStreptococcus suisSerotype 2 InfectionSemantic proximity Read ↗

Immunological characteristics and response to lipopolysaccharide of mouse lines selectively bred with natural and acquired immunities.Semantic proximity Read ↗

Host Genetic Variations and Sex Differences Potentiate Predisposition, Severity, and Outcomes of Group A Streptococcus-Mediated Necrotizing Soft Tissue Infections.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.