p75-deficient sensory axons are immunoreactive for the glycoprotein L1 in mice overexpressing nerve growth factor.

doi:10.1016/s0006-8993(98)00409-0

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Back to search

Article

p75-deficient sensory axons are immunoreactive for the glycoprotein L1 in mice overexpressing nerve growth factor.

Brain research - 6 Jul 1998

Walsh G S, Petruccelli K, Kawaja M D

Abstract excerpt

Nerve growth factor (NGF) regulates the expression of the glycoprotein L1 among neural cell populations. The purpose of this investigation was to determine whether NGF equally affects the immunolocalization of L1 on both sympathetic and sensory axons, and whether the functional expression of the...

Topics

Animals
Axons
Calcitonin Gene-Related Peptide
Cerebellum
Immunohistochemistry
Leukocyte L1 Antigen Complex
Membrane Glycoproteins
Mice
Mice, Inbred C57BL
Mice, Transgenic
Mutation
Nerve Growth Factors
Neural Cell Adhesion Molecules
Neurons, Afferent
Receptors, Nerve Growth Factor
Rhombencephalon
Tyrosine 3-Monooxygenase

Source

Open in PubMed

Identifiers

PMID: 9666123
DOI: 10.1016/s0006-8993(98)00409-0

Genes

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 30 Aug 2026. Prepared record publication-9666123-2026-08-30.1. Manifest publication-graph-cohort-2026-08-30.1.

Source receipt: PubMed via Helena Literature Corpus, retrieved 30 Aug 2026.

Community interpretation

Discuss the evidence

Ask a question, add context or challenge an interpretation. Community activity is separate from scientific evidence strength.

discussions

replies

Citations encouraged

Claims remain attributable.

No discussion yet

Be the first to add a careful, cited interpretation.

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics