Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Differences between the circadian system of two strains of senescence-accelerated mice (SAM).

Physiology & behavior - 1 Dec 1997

Sánchez-Barceló E J, Megias M, Verduga R, Crespo D

Abstract excerpt

The aim of this study was to look for morphological or functional differences between the circadian system of two substrains of Senescence Accelerated Mice (SAM): senescence-prone (SAMP8; average median survival time = 10.0 months) and senescence-resistant (SAMR1; average median survival time = 18.9 months). Neither the general structure nor the quantitative analysis of the number of VP-positive neurons in...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Aging
Share to Circadian Rhythm
Share to Immunohistochemistry

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Mice, Inbred Strains

Share to Motor Activity

Share to Phenotype

Share to Suprachiasmatic Nucleus

Source

Identifiers

PMID: 9383106
DOI: 10.1016/s0031-9384(97)00208-4

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Differences between the circadian system of two strains of senescence-accelerated mice (SAM).PMID 9383106Read ↗

Senescence-accelerated mouse (SAM): a biogerontological resource in aging research.Semantic proximity Read ↗

Senescence-accelerated mouse (SAM): a novel murine model of senescence.Semantic proximity Read ↗

Neuronal Cell Cycle Re-Entry Markers are Altered in the Senescence Accelerated Mouse P8 (SAMP8)Semantic proximity Read ↗

Changes in oxidative stress parameters and neurodegeneration markers in the brain of the senescence-accelerated mice SAMP-8Semantic proximity Read ↗

Pathological and non-pathological aging, SAMP8 and SAMR1. What do hippocampal neuronal populations tell us?Semantic proximity Read ↗

Frontiers of Model Animals for Neuroscience: Two Prosperous Aging Model Animals for Promoting Neuroscience ResearchSemantic proximity Read ↗

Senescence-accelerated mouse (SAM): a novel murine model of accelerated senescence.Semantic proximity Read ↗

Senescence-Accelerated Mouse (SAM) as an Animal Model of Senile Dementia: Pharmacological, Neurochemical and Molecular Biological ApproachSemantic proximity Read ↗

A novel murine model of aging, Senescence-Accelerated Mouse (SAM)Semantic proximity Read ↗

Neuropathological studies on strains of senescence-accelerated mice (SAM) with age-related deficits in learning and memory.Semantic proximity Read ↗

Pathobiology of the senescence-accelerated mouse (SAM).Semantic proximity Read ↗

Effects of accelerated senescence on learning and memory, locomotion and anxiety-like behavior in APP/PS1 mouse model of Alzheimer's disease.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.