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Article

Somatic mutational mechanisms involved in intestinal tumor formation in Min mice.

Cancer research - 15 May 1997

Shoemaker A R, Luongo C, Moser A R, Marton L J, Dove W F

Abstract excerpt

We have demonstrated previously that intestinal tumor formation in B6 Min/+ mice is always accompanied by loss of the wild-type adenomatous polyoposis coli (Apc) allele and that intestinal tumor multiplicity in B6 Min/+ mice can be significantly increased by treatment with a single dose of N-ethy...

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PMID: 9157997

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APC

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 4 Sept 2026. Prepared record publication-9157997-2026-09-04.2. Manifest publication-graph-cohort-2026-09-04.2.

Source receipt: PubMed via Helena Literature Corpus, retrieved 4 Sept 2026.

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Somatic mutational mechanisms involved in intestinal tumor formation in Min mice.PMID 9157997Read ↗

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Intestinal tumours induced by the food carcinogen 2-amino-1-methyl-6-phenylimidazo[4,5-b]pyridine in multiple intestinal neoplasia mice have truncation mutations as well as loss of the wild-type Apc(+) allele.Semantic proximity Read ↗

Adenomatous polyposis coli truncation mutations in 2-amino-1-methyl-6-phenylimidazo[4,5-b]pyridine (PhIP)-induced intestinal tumours of multiple intestinal neoplasia mice.Semantic proximity Read ↗

Somatic genetic events linked to the Apc locus in intestinal adenomas of the Min mouse.Semantic proximity Read ↗

Action of Min and Mom1 on neoplasia in ectopic intestinal grafts.Semantic proximity Read ↗

The Min (multiple intestinal neoplasia) mutation: its effect on gut epithelial cell differentiation and interaction with a modifier system.Semantic proximity Read ↗

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