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Article

A kinase-negative mutation of DNA-PK(CS) in equine SCID results in defective coding and signal joint formation.

Journal of immunology (Baltimore, Md. : 1950) - 15 Apr 1997

Shin E K, Perryman L E, Meek K

Abstract excerpt

The equine SCID defect is more severe than its murine counterpart in that SCID foals are incapable of forming either coding or signal joints, whereas SCID mice manifest normal signal joint formation. To determine the basis of this difference and whether DNA-dependent kinase, catalytic subunit (DN...

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PMID: 9103416

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 2 Sept 2026. Prepared record publication-9103416-2026-09-02.1. Manifest publication-graph-cohort-2026-09-02.1.

Source receipt: PubMed via Helena Literature Corpus, retrieved 2 Sept 2026.

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A kinase-negative mutation of DNA-PK(CS) in equine SCID results in defective coding and signal joint formation.PMID 9103416Read ↗

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