Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Male accessory sex organ morphogenesis is altered by loss of function of Hoxd-13.

Developmental dynamics : an official publication of the American Association of Anatomists - 1 Apr 1997

Podlasek C A, Duboule D, Bushman W

Abstract excerpt

The role of the Hox gene Hoxd-13 in postnatal morphogenesis of the male accessory sex organs was examined by correlating the distribution and temporal regulation of expression in the accessory sex organs of postnatal mice with morphologic abnormalities of Hoxd-13-deficient transgenic mice. Previous studies of Hoxd-13 expression in the perinatal period have shown a broad domain of expression in the lower...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Genitalia, Male
Share to Homeodomain Proteins
Share to Mice

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Mice, Transgenic

Share to Morphogenesis

Share to Mutation

Share to Prostate

Share to Transcription Factors

Source

Identifiers

PMID: 9097018
DOI: 10.1002/(SICI)1097-0177(199704)208:4<454::AID-AJA2>3.0.CO;2-H

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Male accessory sex organ morphogenesis is altered by loss of function of Hoxd-13.PMID 9097018Read ↗

Hoxa-13 gene mutation results in abnormal seminal vesicle and prostate development.Semantic proximity Read ↗

Hoxa-10 deficient male mice exhibit abnormal development of the accessory sex organs.Semantic proximity Read ↗

Genitourinary functions of Hoxa13 and Hoxd13.Semantic proximity Read ↗

Loss of Bmp7 and Fgf8 signaling in Hoxa13-mutant mice causes hypospadia.Semantic proximity Read ↗

Gene dosage-dependent effects of the Hoxa-13 and Hoxd-13 mutations on morphogenesis of the terminal parts of the digestive and urogenital tracts.Semantic proximity Read ↗

Hoxb13 is required for normal differentiation and secretory function of the ventral prostate.Semantic proximity Read ↗

Adult Gli2+/–;Gli3Δ699/+ Male and Female Mice Display a Spectrum of Genital MalformationSemantic proximity Read ↗

The Role of Estrogens in Normal and Abnormal Development of the Prostate GlandSemantic proximity Read ↗

Description and genetic mapping of Polypodia: an X-linked dominant mouse mutant with ectopic caudal limbs and other malformations.Semantic proximity Read ↗

Tail gut endoderm and gut/genitourinary/tail development: a new tissue-specific role for Hoxa13.Semantic proximity Read ↗

The mouse Ulnaless mutation deregulates posterior HoxD gene expression and alters appendicular patterning.Semantic proximity Read ↗

Paralogous mouse Hox genes, Hoxa9, Hoxb9, and Hoxd9, function together to control development of the mammary gland in response to pregnancy.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.