Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

The outcome of the parasitic process initiated by Leishmania infantum in laboratory mice: a tissue-dependent pattern controlled by the Lsh and MHC loci.

Journal of immunology (Baltimore, Md. : 1950) - 15 Nov 1996

Leclercq V, Lebastard M, Belkaid Y, Louis J, Milon G

Abstract excerpt

Human visceral leishmaniasis is mainly due to intracellular protozoan parasites of the Leishmania donovani complex, i.e., L. donovani and L. infantum (or L. chagasi). A mouse model has been established to monitor 1) the parasitic process initiated by L. infantum in three tissues they invade, and 2) parameters of the acquired immune response they trigger. Mice congenic at the Lsh locus and mice of inbred strains...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Alleles
Share to Carrier Proteins

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Major Histocompatibility Complex

Share to Membrane Proteins

Share to Mice, Inbred BALB C

Share to Mice, Inbred C57BL

Share to Organ Specificity

Source

Identifiers

PMID: 8906832

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 5 Sept 2026. Manifest publication-graph-cohort-2026-09-05.1.

Source receipt: pubmed, retrieved 5 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

The outcome of the parasitic process initiated by Leishmania infantum in laboratory mice: a tissue-dependent pattern controlled by the Lsh and MHC loci.PMID 8906832Read ↗

Mechanisms of resistance and susceptibility to experimental visceral leishmaniosis: BALB/c mouse versus syrian hamster modelSemantic proximity Read ↗

The fate and persistence of Leishmania major in mice of different genetic backgrounds: an example of exploitation of the immune system by intracellular parasites.Semantic proximity Read ↗

Vaccination with a Leishmania infantum HSP70-II null mutant confers long-term protective immunity against Leishmania major infection in two mice models.Semantic proximity Read ↗

Leishmania major infection of inbred mice: unmasking genetic determinants of infectious diseases.Semantic proximity Read ↗

Study of Leishmania pathogenesis in mice: experimental considerationsSemantic proximity Read ↗

Phenotypical Characterization of Spleen Remodeling in Murine Experimental Visceral Leishmaniasis.Semantic proximity Read ↗

The immune response of inbred laboratory mice to Litomosoides sigmodontis: A route to discovery in myeloid cell biology.Semantic proximity Read ↗

Leishmanial infection: analysis of its first steps. A reviewSemantic proximity Read ↗

The murine model of infection with Leishmania major and its importance for the deciphering of mechanisms underlying differences in Th cell differentiation in mice from different genetic backgrounds.Semantic proximity Read ↗

Leishmania infantum antigens modulate memory cell subsets of liver resident T lymphocyte.Semantic proximity Read ↗

A cytokine network balance influences the fate of <i>Leishmania (Viannia) braziliensis</i> infection in a cutaneous leishmaniasis hamster modelSemantic proximity Read ↗

Studies in the mouse model identify strain variability as a major determinant of disease outcome in Leishmania infantum infection.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.