Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Epigenetic control of a transposon-inactivated gene in Neurospora is dependent on DNA methylation.

Genetics - 1 May 1996

Cambareri E B, Foss H M, Rountree M R, Selker E U, Kinsey J A

Abstract excerpt

An unstable allele of the Neurospora am (GDH) gene resulting from integration of the retrotransposon Tad3-2 into 5' noncoding sequences was found in previous work. We report that reversion to Am+ depends on DNA methylation within and upstream of Tad. Levels of methylation were correlated with the proportion of Am+ conidia, whether the cultures were derived from Am- or Am+ isolates. Reversion to Am+ did not occur...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Alleles
Share to DNA, Fungal
Share to DNA Transposable Elements

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Genes, Fungal

Share to Genetic Markers

Share to Methylation

Share to Phenotype

Share to Restriction Mapping

Source

Open in PubMed Open full text

Identifiers

PMID: 8722769
DOI: 10.1093/genetics/143.1.137
PMCID: PMC1207247

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Epigenetic control of a transposon-inactivated gene in Neurospora is dependent on DNA methylation.PMID 8722769Read ↗

The DMM complex prevents spreading of DNA methylation from transposons to nearby genes in Neurospora crassa.Semantic proximity Read ↗

DNA methylation is independent of RNA interference in Neurospora.Semantic proximity Read ↗

Abnormal chromosome behavior in Neurospora mutants defective in DNA methylation.Semantic proximity Read ↗

Mutations in the dim-1 gene of Neurospora crassa reduce the level of DNA methylation.Semantic proximity Read ↗

dim-2 encodes a DNA methyltransferase responsible for all known cytosine methylation in Neurospora.Semantic proximity Read ↗

Relationship of vector insert size to homologous integration during transformation of Neurospora crassa with the cloned am (GDH) gene.Semantic proximity Read ↗

Short TpA-rich segments of the zeta-eta region induce DNA methylation in Neurospora crassa.Semantic proximity Read ↗

Small interfering RNAs that trigger posttranscriptional gene silencing are not required for the histone H3 Lys9 methylation necessary for transgenic tandem repeat stabilization in Neurospora crassa.Semantic proximity Read ↗

Is the fungus Magnaporthe losing DNA methylation?Semantic proximity Read ↗

A targeted-replacement system for identification of signals for de novo methylation in Neurospora crassa.Semantic proximity Read ↗

DNA methylation associated with repeat-induced point mutation in Neurospora crassa.Semantic proximity Read ↗

The post-transcriptional gene silencing machinery functions independently of DNA methylation to repress a LINE1-like retrotransposon in Neurospora crassa.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.