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Article

Mos and the cell cycle: the molecular basis of the transformed phenotype.

Current opinion in genetics & development - 1 Feb 1993

Yew N, Strobel M, Vande Woude G F

Abstract excerpt

The product of the mos proto-oncogene is a serine/threonine kinase that is expressed at high levels in germ cells. Mos is a regulator of meiotic maturation, and is required for the initiation and progression of oocyte meiotic maturation that leads to the production of unfertilized eggs. Mos is also a component of cytostatic factor, an activity that is believed to arrest oocyte maturation at meiotic metaphase II....

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Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

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Source

Identifiers

PMID: 8384034
DOI: 10.1016/s0959-437x(05)80336-3

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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Mos and the cell cycle: the molecular basis of the transformed phenotype.PMID 8384034Read ↗

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MOS mutation causes female infertility with large polar body oocytes.Semantic proximity Read ↗

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Meiotic abnormalities of c-mos knockout mouse oocytes: activation after first meiosis or entrance into third meiotic metaphase.Semantic proximity Read ↗

MEIOSIN Directs the Switch from Mitosis to Meiosis in Mammalian Germ CellsSemantic proximity Read ↗

The spindle checkpoint kinase bub1 and cyclin e/cdk2 both contribute to the establishment of meiotic metaphase arrest by cytostatic factor.Semantic proximity Read ↗

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Parthenogenetic development of Mos-deficient mouse oocytes.Semantic proximity Read ↗

DOC1R: a MAP kinase substrate that control microtubule organization of metaphase II mouse oocytesSemantic proximity Read ↗

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