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Article

Eliminating c-fos as a candidate gene for early-onset familial Alzheimer's disease.

Neurology - 1 Sept 1994

Morris S W, St Clair D M

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PMID: 7936314
DOI: 10.1212/wnl.44.9.1762-a

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Related research

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Research trail→

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Eliminating c-fos as a candidate gene for early-onset familial Alzheimer's disease.PMID 7936314Read ↗

The c-fos gene and early-onset familial Alzheimer's disease.Semantic proximity Read ↗

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Increased Immunoreactivity for Jun- and Fos-Related Proteins in Alzheimer's Disease: Association with PathologySemantic proximity Read ↗

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The transcription factor Fos: a Janus-type regulator in health and disease.Semantic proximity Read ↗

Differential Induction of Immediate Early Gene Proteins in Cultured Neurons by β‐Amyloid (Aβ): Association of c‐Jun with Aβ‐Induced ApoptosisSemantic proximity Read ↗

Generation of c-Fos knockout rats, and observation of their phenotype.Semantic proximity Read ↗

c-fos regulates neuronal excitability and survival.Semantic proximity Read ↗

Aggregated Amyloid-β Protein Induces Cortical Neuronal Apoptosis and Concomitant “Apoptotic” Pattern of Gene InductionSemantic proximity Read ↗

ΔFosB Regulates Gene Expression and Cognitive Dysfunction in a Mouse Model of Alzheimer’s DiseaseSemantic proximity Read ↗

Growth of peripheral and central nervous system tumors is supported by cytoplasmic c-Fos in humans and mice.Semantic proximity Read ↗

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