Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

GS domain mutations that constitutively activate T beta R‐I, the downstream signaling component in the TGF‐beta receptor complex.

1 May 1995

Abstract excerpt

No abstract is available from the source.

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Growth Hormone and Insulin-like Growth Factors
Share to Protein Kinase Regulation and GTPase Signaling
Share to TGF-β signaling in diseases

Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 7774578
DOI: 10.1002/j.1460-2075.1995.tb07214.x

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 8 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

GS domain mutations that constitutively activate T beta R‐I, the downstream signaling component in the TGF‐beta receptor complex.PMID 7774578Read ↗

The GS Domain of the Transforming Growth Factor-β Type-I Receptor Is Important in Signal TransductionSemantic proximity Read ↗

Complementation between kinase-defective and activation-defective TGF-beta receptors reveals a novel form of receptor cooperativity essential for signaling.Semantic proximity Read ↗

Novel dominant negative Smad antagonists to TGFbeta signaling.Semantic proximity Read ↗

A Gsalpha mutant designed to inhibit receptor signaling through Gs.Semantic proximity Read ↗

[Functional analysis of transforming growth factor-beta type I receptor].Semantic proximity Read ↗

Going Rogue: Mechanisms, Regulation, and Roles of Mutationally Activated Gα in Human Cancer.Semantic proximity Read ↗

GTPase-inhibiting mutations in the alpha subunit of Gs.Semantic proximity Read ↗

Positive and negative regulation of type II TGF-beta receptor signal transduction by autophosphorylation on multiple serine residues.Semantic proximity Read ↗

TGFBR1 mutations associated with Loeys-Dietz syndrome are inactivating.Semantic proximity Read ↗

Disruption of the interaction between mutationally activated Gαq and Gβγ attenuates aberrant signaling.Semantic proximity Read ↗

Inactivating and activating mutations of the Gs alpha gene.Semantic proximity Read ↗

Ectodomain shedding of TβRIII is required for TβRIII-mediated suppression of TGF-β signaling and breast cancer migration and invasion.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.