Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Acute myeloid leukemia with Inv (16) produces a chimeric transcription factor with a myosin heavy chain tail.

Cold Spring Harbor symposia on quantitative biology - 1 Jan 1994

Liu P, Seidel N, Bodine D, Speck N, Tarlé S, Collins F S

Abstract excerpt

No abstract is available from the source.

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Enhancer Elements, Genetic
Share to 3T3 Cells
Share to Chromosome Aberrations
Share to Chromosomes, Human, Pair 16

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to DNA, Complementary

Share to DNA, Neoplasm

Share to Myosin Heavy Chains

Share to Transcription Factor AP-2

Source

Identifiers

PMID: 7587111
DOI: 10.1101/sqb.1994.059.01.061

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Acute myeloid leukemia with Inv (16) produces a chimeric transcription factor with a myosin heavy chain tail.PMID 7587111Read ↗

The inv(16) fusion protein associates with corepressors via a smooth muscle myosin heavy-chain domain.Semantic proximity Read ↗

[Driver Mutations in Acute Myeloid Leukemia with Inversion of Chromosome 16].Semantic proximity Read ↗

Tissue Specific Requirement for the Inv(16) Oncogene <i>CBFB::MYH11</i> in Acute Myeloid LeukemiaSemantic proximity Read ↗

Pathogenesis of acute myeloid leukaemia and inv(16)(p13;q22): a paradigm for understanding leukaemogenesis?Semantic proximity Read ↗

The inv(16) cooperates with ARF haploinsufficiency to induce acute myeloid leukemia.Semantic proximity Read ↗

Core-Binding Factor Acute Myeloid LeukemiaSemantic proximity Read ↗

Fusion Between Transcription Factor CBFβ/PEBP2β and a Myosin Heavy Chain in Acute Myeloid LeukemiaSemantic proximity Read ↗

FLT3-ITD cooperates with inv(16) to promote progression to acute myeloid leukemia.Semantic proximity Read ↗

Proteogenomic analysis reveals cytoplasmic sequestration of RUNX1 by the acute myeloid leukemia–initiating CBFB::MYH11 oncofusion proteinSemantic proximity Read ↗

Gene-expression profiling identifies distinct subclasses of core binding factor acute myeloid leukemia.Semantic proximity Read ↗

inv(16)/t(16;16) acute myeloid leukemia with non-type A CBFB-MYH11 fusions associate with distinct clinical and genetic features and lack KIT mutations.Semantic proximity Read ↗

Molecular and Clinical Advances in Core Binding Factor Primary Acute Myeloid Leukemia: A Paradigm for Translational Research in Malignant HematologySemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.