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Resistance to activate protein C, the Arg506 to Gln mutation in the factor V gene, and venous thrombosis. Functional tests and DNA-based assays, pros and cons.

Thrombosis and haemostasis - 1 May 1995

Dahlbäck B

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Share to Enzyme Activation
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Share to Factor V Deficiency
Share to Partial Thromboplastin Time

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

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PMID: 7482395

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 8 Sept 2026.

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Resistance to activate protein C, the Arg506 to Gln mutation in the factor V gene, and venous thrombosis. Functional tests and DNA-based assays, pros and cons.PMID 7482395Read ↗

Inherited resistance to activated protein C, a major basis of venous thrombosis, is caused by deficient anticoagulant cofactor function of factor V.Semantic proximity Read ↗

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Resistance to activated protein C due to factor V R506Q mutation as a cause of venous thrombosis.Semantic proximity Read ↗

Laboratory Testing for the Evaluation of Phenotypic Activated Protein C Resistance.Semantic proximity Read ↗

Factor V mutations in Iranian patients with activated protein C resistance and venous thrombosis.Semantic proximity Read ↗

Activated protein C resistance (FVLeiden) and thrombosis: factor V mutations causing hypercoagulable statesSemantic proximity Read ↗

Activated protein C resistance and factor V Leiden: a review.Semantic proximity Read ↗

Protein C deficiency: from gene defects to disease.Semantic proximity Read ↗

Predisposition to thrombosis by a factor V mutation causing hereditary resistance to activated protein C.Semantic proximity Read ↗

Screening for thrombophilia: a laboratory perspective.Semantic proximity Read ↗

Molecular genetics of thrombophilia: factor V gene mutation causing resistance to activated protein C as a basis of the hypercoagulable state.Semantic proximity Read ↗

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