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Article

Molecular genetics of thrombophilia: factor V gene mutation causing resistance to activated protein C as a basis of the hypercoagulable state.

The Journal of laboratory and clinical medicine - 1 May 1995

Dahlbäck B

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Share to Blood Coagulation
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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

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PMID: 7738422

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Molecular genetics of thrombophilia: factor V gene mutation causing resistance to activated protein C as a basis of the hypercoagulable state.PMID 7738422Read ↗

Predisposition to thrombosis by a factor V mutation causing hereditary resistance to activated protein C.Semantic proximity Read ↗

Activated protein C resistance (FVLeiden) and thrombosis: factor V mutations causing hypercoagulable statesSemantic proximity Read ↗

Inherited resistance to activated protein C, a major basis of venous thrombosis, is caused by deficient anticoagulant cofactor function of factor V.Semantic proximity Read ↗

Resistance to activated protein C caused by a factor V gene mutation.Semantic proximity Read ↗

Factor V mutations in Iranian patients with activated protein C resistance and venous thrombosis.Semantic proximity Read ↗

Activated protein C resistance: from phenotype to genotype and clinical practice.Semantic proximity Read ↗

Resistance to activated protein C due to factor V R506Q mutation as a cause of venous thrombosis.Semantic proximity Read ↗

Resistance to activated protein C: a common inherited cause of venous thrombosis.Semantic proximity Read ↗

Thrombophilia: a genetic predisposition to thrombosis.Semantic proximity Read ↗

Factor V Leiden Mutation and the Risks for Thromboembolic Disease: A Clinical PerspectiveSemantic proximity Read ↗

Brief report: variability of thrombosis among homozygous siblings with resistance to activated protein C due to an Arg-->Gln mutation in the gene for factor V.Semantic proximity Read ↗

Molecular pathophysiology of thrombotic states and their impact to laboratory diagnostics.Semantic proximity Read ↗

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