Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Alterations in mucosa branched N -glycans lead to dysbiosis and downregulation of ILC3: a key driver of intestinal inflammation

7 Feb 2025

Abstract excerpt

The perturbation of the symbiotic relationship between microbes and intestinal immune system contributes to gut inflammation and Inflammatory Bowel Disease (IBD) development. The host mucosa glycans (glycocalyx) creates a major biological interface between gut microorganisms and host immunity that remains ill-defined. Glycans are essential players in IBD immunopathogenesis, even years before disease onset....

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Eosinophilic Esophagitis
Share to Helicobacter pylori-related gastroenterology studies
Share to IL-33, ST2, and ILC Pathways

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Source

Identifiers

PMID: 39918275
DOI: 10.1080/19490976.2025.2461210

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 8 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Alterations in mucosa branched N -glycans lead to dysbiosis and downregulation of ILC3: a key driver of intestinal inflammationPMID 39918275Read ↗

The role of glycans in health and disease: Regulators of the interaction between gut microbiota and host immune systemSemantic proximity Read ↗

Glycans as critical regulators of gut immunity in homeostasis and diseaseSemantic proximity Read ↗

The immune interactions of gut glycans and microbiota in health and diseaseSemantic proximity Read ↗

Role of aberrant IgG glycosylation in the pathogenesis of inflammatory bowel diseaseSemantic proximity Read ↗

Gut Microbiota Serves as a Crucial Independent Biomarker in Inflammatory Bowel Disease (IBD)Semantic proximity Read ↗

Arthritis-Associated Inflammation Remodels Colonic O-GlycosylationSemantic proximity Read ↗

Inflammatory bowel disease associates with proinflammatory potential of the immunoglobulin G glycome.Semantic proximity Read ↗

Epithelial glycosylation in gut homeostasis and inflammation.Semantic proximity Read ↗

Loss of intestinal O-glycans promotes spontaneous duodenal tumors.Semantic proximity Read ↗

Decoding gut microbiome alterations in celiac disease: Implications for pathogenesis and treatment.Semantic proximity Read ↗

Development of Inflammatory Bowel Disease is Linked to a Longitudinal Restructuring of the Gut Metagenome in MiceSemantic proximity Read ↗

Mucin function in inflammatory bowel disease: an update.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.