Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

LncRNA MIR210HG promotes phenotype switching of pulmonary arterial smooth muscle cells through autophagy-dependent ferroptosis pathway.

Apoptosis : an international journal on programmed cell death - 1 Oct 2024

Wang Enze, Zhang Binbin, Huang Ling, Li Pulin, Han Rui, Zhou Sijing, Zeng Daxiong, Wang Ran

Abstract excerpt

Hypoxic pulmonary hypertension (HPH) is a pathophysiological syndrome in which pulmonary vascular pressure increases under hypoxic stimulation and there is an urgent need to develop emerging therapies for the treatment of HPH. LncRNA MIR210HG is a long non-coding RNA closely related to hypoxia and has been widely reported in a variety of tumor diseases. But its mechanism in hypoxic pulmonary hypertension is not...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Basic Helix-Loop-Helix Transcription Factors
Share to Cell Hypoxia

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Ferroptosis

Share to Hypertension, Pulmonary

Share to Hypoxia

Share to Myocytes, Smooth Muscle

Share to Pulmonary Artery

Share to STAT3 Transcription Factor

Source

Identifiers

PMID: 38635022
DOI: 10.1007/s10495-024-01963-4

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 5 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

LncRNA MIR210HG promotes phenotype switching of pulmonary arterial smooth muscle cells through autophagy-dependent ferroptosis pathway.PMID 38635022Read ↗

Research progress on the mechanism of phenotypic transformation of pulmonary artery smooth muscle cells induced by hypoxiaSemantic proximity Read ↗

New insights into the pathology of pulmonary hypertension: implication of the miR-210/ISCU1/2/Fe-S axis.Semantic proximity Read ↗

An Overview of miRNAs Involved in PASMC Phenotypic Switching in Pulmonary Hypertension.Semantic proximity Read ↗

Overexpressed lncRNA AC068039.4 contributes to proliferation and cell cycle progression of pulmonary artery smooth muscle cells via sponging miR-26a-5p/TRPC6 in hypoxic pulmonary arterial hypertensionSemantic proximity Read ↗

CircLMBR1 inhibits phenotypic transformation of hypoxia-induced pulmonary artery smooth muscle via the splicing factor PUF60.Semantic proximity Read ↗

miR-206 regulates hypoxia-induced mitophagy and phenotypic remodeling in yak pulmonary artery smooth muscle cells.Semantic proximity Read ↗

Exosomes derived from hypoxic alveolar epithelial cells promote the phenotypic transformation of pulmonary artery smooth muscle cells via the Rap1 pathway.Semantic proximity Read ↗

Long Noncoding RNA-Maternally Expressed Gene 3 Contributes to Hypoxic Pulmonary HypertensionSemantic proximity Read ↗

Updated perspective of EPAS1 and the role in pulmonary hypertensionSemantic proximity Read ↗

Super Enhancer-driven LncRNA UNC5B-AS1 Inhibits Inflammatory Phenotypic Transition in Smooth Muscle Cells via Lactylation ModificationSemantic proximity Read ↗

Hypoxia-induced miR-182-5p regulates vascular smooth muscle cell phenotypic switch by targeting RGS5.Semantic proximity Read ↗

MiR-20a regulates the PRKG1 gene by targeting its coding region in pulmonary arterial smooth muscle cells.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.