Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

ANKRD22 promotes resolution of psoriasiform skin inflammation by antagonizing NIK-mediated IL-23 production

7 Mar 2024

Abstract excerpt

No abstract is available from the source.

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Dermatology and Skin Diseases
Share to Immune Response and Inflammation
Share to Psoriasis: Treatment and Pathogenesis

Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 38454607
DOI: 10.1016/j.ymthe.2024.03.007

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 8 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

ANKRD22 promotes resolution of psoriasiform skin inflammation by antagonizing NIK-mediated IL-23 productionPMID 38454607Read ↗

Targeting Interleukin-22 in PsoriasisSemantic proximity Read ↗

NOD-like receptor signaling and inflammasome-related pathways are highlighted in psoriatic epidermisSemantic proximity Read ↗

NLRP1 Inflammasome Activation in Keratinocytes: Increasing Evidence of Important Roles in Inflammatory Skin Diseases and ImmunitySemantic proximity Read ↗

Psoriasiform dermatitis is driven by IL-36–mediated DC-keratinocyte crosstalkSemantic proximity Read ↗

Keratinocyte Expression of A20/TNFAIP3 Controls Skin Inflammation Associated with Atopic Dermatitis and PsoriasisSemantic proximity Read ↗

LCN2 Mediates Skin Inflammation in Psoriasis through the SREBP2‒NLRC4 AxisSemantic proximity Read ↗

Autoinflammatory keratinization diseases: An emerging concept encompassing various inflammatory keratinization disorders of the skin.Semantic proximity Read ↗

A Comprehensive Analysis of Pattern Recognition Receptors in Normal and Inflamed Human Epidermis: Upregulation of Dectin-1 in PsoriasisSemantic proximity Read ↗

IL-22 Down-Regulates Cx43 Expression and Decreases Gap Junctional Intercellular Communication by Activating the JNK Pathway in PsoriasisSemantic proximity Read ↗

Keratinocytes contribute intrinsically to psoriasis upon loss of Tnip1 functionSemantic proximity Read ↗

IL-22RA2 Is a SMAD7 Target Mediating the Alleviation of Dermatitis and Psoriatic Phenotypes in Mice.Semantic proximity Read ↗

IL‐22 regulates the expression of genes responsible for antimicrobial defense, cellular differentiation, and mobility in keratinocytes: a potential role in psoriasisSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.