Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Knockdown of RTEL1 Alleviates Chronic Obstructive Pulmonary Disease by Modulating M1, M2 Macrophage Polarization and Inflammation

29 Feb 2024

Abstract excerpt

Chronic obstructive pulmonary disease (COPD) is a common chronic disease characterized by airflow obstruction, which seriously threatens people's health. The COPD mouse model was established with cigarette smoke induction. Hematoxylin-eosin staining and Masson staining were carried out to observe the pathological changes of lung tissues in COPD mice. RTEL1 was silenced in COPD mice, and immunohistochemistry was...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Chronic Obstructive Pulmonary Disease (COPD) Research
Share to Immune cells in cancer
Share to MicroRNA in disease regulation

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 38420994
DOI: 10.1080/15412555.2024.2316607

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 6 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Knockdown of RTEL1 Alleviates Chronic Obstructive Pulmonary Disease by Modulating M1, M2 Macrophage Polarization and InflammationPMID 38420994Read ↗

Characteristics and potential role of M2 macrophages in COPD.Semantic proximity Read ↗

Molecular changes underlying pulmonary emphysema and chronic bronchitis in Chronic Obstructive Pulmonary Disease: An updated review.Semantic proximity Read ↗

Macrophage Polarization and Functions in Pathogenesis of Chronic Obstructive Pulmonary DiseaseSemantic proximity Read ↗

ZBP1 aggravates acute lung injury in mice by promoting the macrophage inflammatory phenotype.Semantic proximity Read ↗

Targeting BRD4 Ameliorates Experimental Emphysema by Disrupting Super-Enhancer in Polarized Alveolar MacrophageSemantic proximity Read ↗

S1P/HDAC1 Signaling Regulates Macrophage Polarization in Patients with Chronic Obstructive Pulmonary DiseaseSemantic proximity Read ↗

Macrophage Polarization: An Important Candidate Regulator for Lung Diseases.Semantic proximity Read ↗

Roles of Macrophage Polarization and Macrophage-Derived miRNAs in Pulmonary FibrosisSemantic proximity Read ↗

TROP2 promotes PINK1-mediated mitophagy and apoptosis to accelerate the progression of senile chronic obstructive pulmonary disease by up-regulating DRP1 expressionSemantic proximity Read ↗

Current views in chronic obstructive pulmonary disease pathogenesis and managementSemantic proximity Read ↗

Abnormal M1/M2 macrophage phenotype profiles in the small airway wall and lumen in smokers and chronic obstructive pulmonary disease (COPD).Semantic proximity Read ↗

Single-Cell RNA Sequencing Provides New Insights into Therapeutic Roles of Thyroid Hormone in Idiopathic Pulmonary FibrosisSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.