Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Cerebrovascular response to exercise interacts with individual genotype and amyloid-beta deposition to influence response inhibition with aging.

Neurobiology of aging - 1 Jun 2022

Palmer Jacqueline A, Kaufman Carolyn S, Vidoni Eric D, Honea Robyn A, Burns Jeffrey M, Billinger Sandra A

Abstract excerpt

The etiology of cognitive dysfunction associated with Alzheimer's disease (AD) and dementia is multifactorial. Yet, mechanistic interactions among key neurobiological factors linked to AD pathology are unclear. This study tested the effect of interactions between cerebrovascular function, individual genotype, and structural brain pathology on response inhibition performance, an early and sensitive indicator of...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Aging
Share to Alzheimer Disease

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Amyloid beta-Peptides

Share to Apolipoprotein E4

Share to Cognition

Share to Exercise

Share to Positron-Emission Tomography

Source

Open in PubMed Open full text

Identifiers

PMID: 35344819
DOI: 10.1016/j.neurobiolaging.2022.02.014
PMCID: PMC9731173

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 5 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Cerebrovascular response to exercise interacts with individual genotype and amyloid-beta deposition to influence response inhibition with aging.PMID 35344819Read ↗

Cerebrovascular response to exercise interacts with individual genotype and amyloid-beta deposition to influence response inhibition with agingSemantic proximity Read ↗

APOE4 carriers resistant to cognitive decline show unique relationships between cerebrovascular response to exercise and dual-task cognitive-balance performanceSemantic proximity Read ↗

Dementia Risk and Dynamic Response to Exercise: A non-randomized clinical trialSemantic proximity Read ↗

Cerebral Vasoreactivity, Apolipoprotein E, and the Risk of Dementia: A Population-Based Study.Semantic proximity Read ↗

APOE genotype modifies the association between central arterial stiffening and cognition in older adults.Semantic proximity Read ↗

Cerebrovascular regulation dynamics and Alzheimer's neuroimaging phenotypes.Semantic proximity Read ↗

APOE4 carriers display loss of anticipatory cerebrovascular regulation across the Alzheimer's disease continuum.Semantic proximity Read ↗

Reduced cerebrovascular reactivity in young adults carrying the APOE ε4 allele.Semantic proximity Read ↗

Stressor-evoked brain activity, cardiovascular reactivity, and subclinical atherosclerosis in midlife adultsSemantic proximity Read ↗

APOE4 carriers display loss of anticipatory cerebral vascular regulation over AD progressionSemantic proximity Read ↗

Dementia Risk and Dynamic Response to Exercise: Methodology for an Acute Exericise Clinical TrialSemantic proximity Read ↗

Acute Exercise Effect on Neurocognitive Function Among Cognitively Normal Late-Middle-Aged Adults With/Without Genetic Risk of AD: The Moderating Role of Exercise Volume and APOE Genotype.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.