Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Elevated plasma levels of CXCL16 in severe COVID-19 patients

30 Jan 2022

Abstract excerpt

Genome-wide association studies have recently identified 3p21.31, with lead variant pointing to the CXCR6 gene, as the strongest thus far reported susceptibility risk locus for severe manifestation of COVID-19. In order the determine its role, we measured plasma levels of Chemokine (C-X-C motif) ligand 16 (CXCL16) in the plasma of COVID-19 hospitalized patients. CXCL16 interacts with CXCR6 promoting chemotaxis or...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to COVID-19 Clinical Research Studies
Share to Immune responses and vaccinations
Share to SARS-CoV-2 and COVID-19 Research

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 35121493
DOI: 10.1016/j.cyto.2022.155810

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Elevated plasma levels of CXCL16 in severe COVID-19 patientsPMID 35121493Read ↗

Elevated plasma levels of CXCL16 in severe COVID-19 patientsSemantic proximity Read ↗

The CXCR6/CXCL16 axis links inflamm-aging to disease severity in COVID-19 patientsSemantic proximity Read ↗

Single cell sequencing analysis uncovers genetics-influenced CD16+monocytes and memory CD8+T cells involved in severe COVID-19Semantic proximity Read ↗

Effect of Chemokine Gene Variants on Covid-19 Disease Severity.Semantic proximity Read ↗

Molecular Determination of Tumor Necrosis Factor-alpha, Interleukin-8, Interleukin-10, and C-X-C Chemokine Receptor-2 Genetic Variations and their Association with Disease Susceptibility and Mortality in COVID-19 PatientsSemantic proximity Read ↗

Integrating single-cell sequencing data with GWAS summary statistics reveals CD16+monocytes and memory CD8+T cells involved in severe COVID-19.Semantic proximity Read ↗

Association of <i>CXCR6</i> with COVID-19 severity: Delineating the host genetic factors in transcriptomic regulationSemantic proximity Read ↗

Transcriptional Changes in CD16+ Monocytes May Contribute to the Pathogenesis of COVID-19Semantic proximity Read ↗

Common variants at 21q22.3 locus influence<i>MX1</i>gene expression and susceptibility to severe COVID-19Semantic proximity Read ↗

Regulatory Noncoding and Predicted Pathogenic Coding Variants of CCR5 Predispose to Severe COVID-19.Semantic proximity Read ↗

Impact of IL-6 rs1800795 and IL-17A rs2275913 gene polymorphisms on the COVID-19 prognosis and susceptibility in a sample of Iranian patients.Semantic proximity Read ↗

Severe COVID-19 associated variants linked to chemokine receptor gene control in monocytes and macrophagesSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.