Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

miR‐24 and its target gene Prdx6 regulate viability and senescence of myogenic progenitors during aging

24 Sept 2021

Abstract excerpt

Satellite cell-dependent skeletal muscle regeneration declines during aging. Disruptions within the satellite cells and their niche, together with alterations in the myofibrillar environment, contribute to age-related dysfunction and defective muscle regeneration. In this study, we demonstrated an age-related decline in satellite cell viability and myogenic potential and an increase in ROS and cellular...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Muscle Physiology and Disorders
Share to Telomeres, Telomerase, and Senescence
Share to Tissue Engineering and Regenerative Medicine

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Source

Identifiers

PMID: 34560818
DOI: 10.1111/acel.13475

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 9 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

miR‐24 and its target gene Prdx6 regulate viability and senescence of myogenic progenitors during agingPMID 34560818Read ↗

miR-24:Prdx6 interactions regulate oxidative stress and viability of myogenic progenitors during ageingSemantic proximity Read ↗

Cellular Senescence in the Regenerative Niche Hampers Skeletal Muscle RepairSemantic proximity Read ↗

Muscle stem cells in Duchenne muscular dystrophy exhibit molecular impairments and altered cell fate trajectories impacting regenerative capacitySemantic proximity Read ↗

Muscle stem cells in Duchenne muscular dystrophy exhibit molecular impairments and altered cell fate trajectories impacting regenerative capacitySemantic proximity Read ↗

Muscle-derived stem/progenitor cell dysfunction in Zmpste24-deficient progeroid mice limits muscle regeneration.Semantic proximity Read ↗

Age-related declines in α-Klotho drive progenitor cell mitochondrial dysfunction and impaired muscle regenerationSemantic proximity Read ↗

Cellular senescence-mediated exacerbation of Duchenne muscular dystrophy.Semantic proximity Read ↗

Restoration of CPEB4 prevents muscle stem cell senescence during agingSemantic proximity Read ↗

Role of miR‐200c in Myogenic Differentiation Impairment via p66Shc: Implication in Skeletal Muscle Regeneration of Dystrophic mdx MiceSemantic proximity Read ↗

Regulation of DMD pathology by an ankyrin-encoded miRNASemantic proximity Read ↗

Macrophage Subpopulation Promotes Skeletal Muscle Regeneration Through HGF / MET Signaling‐Mediated Skeletal Muscle Stem Cell ProliferationSemantic proximity Read ↗

Molecular basis of the myogenic profile of aged human skeletal muscle satellite cells during differentiationSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.